Evidence mapPaperPMID 34071103Full record

ReviewLife (Basel, Switzerland)2021

Antiplatelet Effects of PCSK9 Inhibitors in Primary Hypercholesterolemia.

Piotr Pęczek, Mateusz Leśniewski, Tomasz Mazurek, Lukasz Szarpak, Krzysztof J Filipiak, Aleksandra Gąsecka

Open access · goldAbstract readReview
In one paragraph

Review in Life (Basel, Switzerland), 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
4.0field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

15 citing papers in PubMed, 19 citations in OpenAlex.

  1. Trial
  2. Article
  3. Review
  4. Review
  5. Review
  6. Lipid-Lowering Therapy after Acute Coronary Syndrome.Journal of clinical medicine · 2024
    Review
  7. Article
  8. Article
  9. The Anti-Thrombotic Effects of PCSK9 Inhibitors.Pharmaceuticals (Basel, Switzerland) · 2023
    Review
  10. Review
  11. Article
  12. Review
  13. Review
  14. Review
  15. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 2 countries.

Piotr Pęczek1st Chair and Department of Cardiology, Medical University of Warsaw, 00-927 Warsaw, Poland.
Mateusz Leśniewski1st Chair and Department of Cardiology, Medical University of Warsaw, 00-927 Warsaw, Poland.ORCID 0000-0002-7914-2022
Tomasz Mazurek1st Chair and Department of Cardiology, Medical University of Warsaw, 00-927 Warsaw, Poland.ORCID 0000-0002-3693-8741
Lukasz SzarpakDepartment of Research Outcomes, Maria Sklodowska-Curie Medical Academy in Warsaw, 03-411 Warsaw, Poland.ORCID 0000-0002-0973-5455
Krzysztof J Filipiak1st Chair and Department of Cardiology, Medical University of Warsaw, 00-927 Warsaw, Poland.ORCID 0000-0002-6563-0877
Aleksandra Gąsecka1st Chair and Department of Cardiology, Medical University of Warsaw, 00-927 Warsaw, Poland.ORCID 0000-0001-5083-7587
Medical University of Warsaw · PLMedical University of Białystok · PL

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Proprotein convertase subtilisin-kexin type 9 (PCSK9) inhibitors are a novel group of hypolipidemic drugs that are recommended particularly for high-risk hypercholesterolemia patients, including those with primary hypercholesterolemia (PH), where lifelong exposure to high low-density lipoprotein (LDL) cholesterol levels results in an elevated risk of atherosclerosis at an early age. The onset and progression of atherosclerosis is significantly influenced by activated platelets. Oxidized LDL influences platelet activation by interacting with their surface receptors and remodeling the composition of their cell membrane. This results in platelet aggregation, endothelial cell activation, promotion of inflammation and oxidative stress, and acceleration of lipid accumulation in atherosclerotic plaques. PCSK9 inhibitors reduce platelet activation by both significantly lowering LDL levels and reducing the LDL receptor-mediated activation of platelets by PCSK9. They also work synergistically with other hypolipidemic and antithrombotic drugs, including statins, ezetimibe, acetylsalicylic acid, clopidogrel, and ticagrelor, which enhances their antiplatelet and LDL-lowering effects. In this review, we summarize the currently available evidence on platelet hyperreactivity in PH, the effects of PCSK9 inhibitors on platelets, and their synergism with other drugs used in PH therapy.

Indexed as

atherosclerosisLDL-cholesterolPCSK9 inhibitorsplateletsprimary hypercholesterolemiatreatment

Identifiers

PMID34071103
PMCPMC8224623
OpenAlexW3164626826

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.