Evidence mapPaperPMID 34097014Full record

ArticleRheumatology (Oxford, England)2022

Effect of the phosphodiesterase 4 inhibitor apremilast on cardiometabolic outcomes in psoriatic disease-results of the Immune Metabolic Associations in Psoriatic Arthritis study.

Lyn D Ferguson, Susanne Cathcart, Dominic Rimmer, Gary Semple, Katriona Brooksbank, Caron Paterson, Rosemary Brown, John Harvie, Xuan Gao, Aleksandra Radjenovic and 4 more

Open access · hybridAbstract read
In one paragraph

Article in Rheumatology (Oxford, England), 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 23 papers.

0numbers the graph read from it
0cells of the map it votes in
23citing papers in PubMed
4.6field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

23 citing papers in PubMed, 39 citations in OpenAlex.

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  18. Acute PDE4 Inhibition Induces a Transient Increase in Blood Glucose in Mice.International journal of molecular sciences · 2023
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

14 authors at 4 institutions in 2 countries.

Lyn D FergusonInstitute of Cardiovascular and Medical Sciences, University of Glasgow.ORCID 0000-0002-6136-8349
Susanne CathcartGlasgow Clinical Research Facility, Glasgow Royal Infirmary.
Dominic RimmerGlasgow Clinical Research Facility, Glasgow Royal Infirmary.
Gary SempleGlasgow Clinical Research Facility, Glasgow Royal Infirmary.
Katriona BrooksbankInstitute of Cardiovascular and Medical Sciences, University of Glasgow.
Caron PatersonInstitute of Infection, Immunity, and Inflammation, University of Glasgow, Glasgow.
Rosemary BrownInstitute of Cardiovascular and Medical Sciences, University of Glasgow.
John HarvieRaigmore Hospital, Inverness, UK.
Xuan GaoInstitute of Cardiovascular and Medical Sciences, University of Glasgow.
Aleksandra RadjenovicInstitute of Cardiovascular and Medical Sciences, University of Glasgow.
Paul WelshInstitute of Cardiovascular and Medical Sciences, University of Glasgow.
Iain B McInnesInstitute of Infection, Immunity, and Inflammation, University of Glasgow, Glasgow.
Naveed SattarInstitute of Cardiovascular and Medical Sciences, University of Glasgow.
Stefan SiebertInstitute of Infection, Immunity, and Inflammation, University of Glasgow, Glasgow.ORCID 0000-0002-1802-7311
University of Glasgow · GBGlasgow Royal Infirmary · GBInstitute of Infection and Immunity · CARaigmore Hospital · GB

Funding

British Heart Foundation RE/13/5/30177
6 · The paper itself

Abstract

objectivesStudies have suggested phosphodiesterase 4 (PDE4) inhibition may be associated with weight loss and other cardiometabolic benefits. We evaluated the effect of the PDE4 inhibitor apremilast on body weight and composition, glucose homeostasis, lipid profiles and vascular function in psoriatic disease and whether weight change correlated with therapeutic response.

methodsWe conducted a prospective, open-label study (Immune Metabolic Associations in Psoriatic Arthritis) of adults receiving apremilast 30 mg as part of routine care for PsA and/or psoriasis. Cardiometabolic, anthropometric and disease activity assessments were performed at baseline (pre-apremilast) and at months 1, 3 and 6 of apremilast treatment in 60 patients. A subgroup underwent further assessment of endothelial function, body composition and adipocyte morphology.

resultsIn patients (median age 54.5 years, 63% women, median BMI 33.2 kg/m2), apremilast was associated with a mean weight loss of 2.2 kg (95% CI 1.4, 3.0; P < 0.001) and a mean BMI decrease of 0.8 kg/m2 (95% CI 0.5, 1.2; P < 0.001) after 6 months of treatment. Body composition analysis demonstrated a reduction in total abdominal fat [mean decrease 0.52 L (95% CI 0.08, 0.96), P = 0.022], principally subcutaneous adipose tissue [mean decrease 0.37 L (95% CI 0.05, 0.68), P = 0.022]. There was no change in adipocyte diameter, haemoglobin A1c, lipid, glucagon-like peptide-1 or vascular function. Psoriatic disease activity improved with apremilast, although this was not correlated with weight change.

conclusionFollowing apremilast treatment, we observed weight loss, principally abdominal subcutaneous fat, and improvement in psoriatic disease activity. The latter was independent of weight change, suggesting apremilast likely acts through direct immunological mechanisms.

Indexed as

Cardiometabolic Risk FactorsAdultArthritis, PsoriaticBody Fat DistributionFemaleHumansMaleMiddle AgedPhosphodiesterase 4 InhibitorsProspective StudiesPsoriasisThalidomideWeight LossapremilastPhosphodiesterase 4 InhibitorsThalidomideadipose tissueapremilastectopic fatmetabolicPDE4 inhibitionpsoriatic diseasevascularweight

Identifiers

PMID34097014
PMCPMC8889283
OpenAlexW3169197984

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.