Evidence map›Paper›PMID 34185201›Full record

ReviewInflammopharmacology2021

Molecular and pathobiological involvement of fetuin-A in the pathogenesis of NAFLD.

Ojus Sardana, Ravi Goyal, Onkar Bedi

Abstract readReview
PubMed Publisher
In one paragraph

Review in Inflammopharmacology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed, 1 pooled it
3.3field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

20 citing papers in PubMed, 1 synthesis or guideline pooled it, 30 citations in OpenAlex.

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  20. The structure, biosynthesis, and biological roles of fetuin-A: A review.Frontiers in cell and developmental biology · 2022
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Ojus SardanaChitkara College of Pharmacy, Chitkara University, Rajpura, Punjab, India.
Ravi GoyalChitkara College of Pharmacy, Chitkara University, Rajpura, Punjab, India.
Onkar BediChitkara College of Pharmacy, Chitkara University, Rajpura, Punjab, India. onkar.bedi@chitkara.edu.in.ORCID http://orcid.org/0000-0003-4724-366X
Chitkara University · IN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The liver acts as a manufacturing unit for the production of fetuin-A, which is essential for various physiological characteristics. Scientific research has shown that a moderate upward push in fetuin-A serum levels is associated with a confirmed non-alcoholic fatty liver disease (NAFLD) diagnosis. Fetuin-A modulation is associated with a number of pathophysiological variables that cause liver problems, including insulin receptor signaling deficiencies, adipocyte dysfunction, hepatic inflammation, fibrosis, triacylglycerol production, macrophage invasion, and TLR4 activation. The focus of the present review is on the various molecular pathways, and genetic relevance of mRNA expression of fetuin-A which is correlated with progression of NAFLD. The other major area of exploration in the present review is based on the new targets for the modulation of fetuin-A, like calorie restriction and novel pharmacological agents, such as rosuvastatin, metformin, and pioglitazone which are successfully implicated in the management of various liver-related complications.

Indexed as

Adiponectinalpha-2-HS-GlycoproteinAnimalsAnti-Inflammatory AgentsCaloric RestrictionHumansHypoglycemic AgentsInflammation MediatorsInsulin ResistanceLiverMetforminNon-alcoholic Fatty Liver DiseaseAdiponectinAHSG protein, humanalpha-2-HS-GlycoproteinAnti-Inflammatory AgentsHypoglycemic AgentsInflammation MediatorsMetforminFetuin-AInflammatoryInsulin resistance (IR)Metabolic disordersNon-alcoholic fatty liver disease (NAFLD)

Identifiers

PMID34185201
OpenAlexW3173287814

What Socratic holds

Texttitle and abstract
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.