Evidence mapPaperPMID 34270402Full record

ArticleRambam Maimonides medical journal2021

Circulating Proprotein Convertase Subtilisin/Kexin Type 9 Levels Predict Future Cardiovascular Event Risks in Hemodialyzed Black African Patients.

François-Pantaléon Musungayi Kajingulu, François Bompeka Lepira, Aliocha Natuhoyila Nkodila, Jean-Robert Rissassy Makulo, Vieux Momeme Mokoli, Pepe Mfutu Ekulu, Justine Busanga Bukabau, Yannick Mayamba Nlandu, Augustin Luzayadio Longo, Nazaire Mangani Nseka and 1 more

Open access · diamondAbstract read
In one paragraph

Article in Rambam Maimonides medical journal, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.6field-weighted citation impact, top 31% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 4 citations in OpenAlex.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 3 institutions in 2 countries.

François-Pantaléon Musungayi KajinguluDepartment of Internal Medicine, Division of Nephrology-Dialysis, University of Kinshasa Hospital, Kinshasa, Democratic Republic of the Congo.
François Bompeka LepiraDepartment of Internal Medicine, Division of Nephrology-Dialysis, University of Kinshasa Hospital, Kinshasa, Democratic Republic of the Congo.
Aliocha Natuhoyila NkodilaFaculty of Family Medicine and Primary Care, Protestant University of Congo, Kinshasa, Democratic Republic of the Congo.
Jean-Robert Rissassy MakuloDepartment of Internal Medicine, Division of Nephrology-Dialysis, University of Kinshasa Hospital, Kinshasa, Democratic Republic of the Congo.
Vieux Momeme MokoliDepartment of Internal Medicine, Division of Nephrology-Dialysis, University of Kinshasa Hospital, Kinshasa, Democratic Republic of the Congo.
Pepe Mfutu EkuluDepartment of Pediatrics, Division of Nephrology-Dialysis, University of Kinshasa Hospital, Kinshasa, Democratic Republic of the Congo.
Justine Busanga BukabauDepartment of Internal Medicine, Division of Nephrology-Dialysis, University of Kinshasa Hospital, Kinshasa, Democratic Republic of the Congo.
Yannick Mayamba NlanduDepartment of Internal Medicine, Division of Nephrology-Dialysis, University of Kinshasa Hospital, Kinshasa, Democratic Republic of the Congo.
Augustin Luzayadio LongoDepartment of Internal Medicine, Division of Nephrology-Dialysis, University of Kinshasa Hospital, Kinshasa, Democratic Republic of the Congo.
Nazaire Mangani NsekaDepartment of Internal Medicine, Division of Nephrology-Dialysis, University of Kinshasa Hospital, Kinshasa, Democratic Republic of the Congo.
Ernest Kiswaya SumailiDepartment of Internal Medicine, Division of Nephrology-Dialysis, University of Kinshasa Hospital, Kinshasa, Democratic Republic of the Congo.
University of Kinshasa · CDPediatrics and Genetics · USUniversité Protestante au Congo · CD

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

CONTEXT AND

objectiveCardiovascular diseases are the leading cause of mortality in patients. In this context, proprotein convertase subtilisin/kexin type 9 (PCSK9) appears to be the new biomarker identified as interfering in lipid homeostasis. This study aimed to investigate the association between PCSK9, dyslipidemia, and future risk of cardiovascular events in a population of black Africans.

methodsA cross-sectional study was conducted between August 2016 and July 2020 in six hemodialysis centers in the city of Kinshasa, Democratic Republic of the Congo. Serum PCSK9 was measured by ELISA; lipid levels of 251 chronic kidney disease grade 5 (CKD G5) hemodialysis patients and the Framingham predictive instrument were used for predicting cardiac events.

resultsTotal cholesterol (TC), low-density lipoprotein cholesterol (LDL-c), and triglycerides (TG) were significantly increased in the tertile with the highest PCSK9. By contrast, high-density lipoprotein cholesterol (HDL-c) was significantly decreased in the same tertile. A strong positive and significant correlation was found between PCSK9 and TC, TG, and LDL-c. Negative and significant correlation was observed between PCSK9 and HDL-c. The levels of PCSK9, smoking, overweight, and atherogenic dyslipidemia were associated with future risks for cardiovascular events in univariate analysis. After adjustment, all these variables persisted as independent determinants of future risk for cardiovascular events. The probability of having a cardiovascular event in this population was independently associated with PCSK9 levels. Compared to the patients in the lowest PCSK9 tertile, patients with PCSK9 levels in the middle (aOR 5.9, 95% CI 2.06-17.3, P<0.001) and highest tertiles (aOR 8.9, 95% CI 3.02-25.08, P<0.001) presented a greater risk of cardiac event.

conclusionIncreased PCSK9 serum levels are associated with higher levels of TC, LDL-c, and TG and lower levels of HDL-c in black African hemodialysis patients. Serum PCSK9 levels in these patients predict increased risk of cardiovascular events, independent of traditional potential confounders.

Identifiers

PMID34270402
PMCPMC8284989
OpenAlexW3177992883

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.