Evidence mapPaperPMID 34299048Full record

ReviewInternational journal of molecular sciences2021

Adipose-Derived Exosomes as Possible Players in the Development of Insulin Resistance.

Arkadiusz Żbikowski, Agnieszka Błachnio-Zabielska, Mauro Galli, Piotr Zabielski

Abstract readReview
In one paragraph

Review in International journal of molecular sciences, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 21 papers.

0numbers the graph read from it
0cells of the map it votes in
21citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

21 citing papers in PubMed.

  1. Review
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  8. Article
  9. Review
  10. Article
  11. Review
  12. Review
  13. The Landscape of Exosomes Biogenesis to Clinical Applications.International journal of nanomedicine · 2024
    Review
  14. Review
  15. Article
  16. Review
  17. Review
  18. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Arkadiusz ŻbikowskiDepartment of Medical Biology, Medical University of Bialystok, 15-089 Białystok, Poland.ORCID 0000-0002-4276-053X
Agnieszka Błachnio-ZabielskaDepartment of Hygiene, Epidemiology and Metabolic Disorders, Medical University of Bialystok, 15-089 Białystok, Poland.ORCID 0000-0002-8055-0576
Mauro GalliDepartment of Medical Biology, Medical University of Bialystok, 15-089 Białystok, Poland.ORCID 0000-0003-3871-8125
Piotr ZabielskiDepartment of Medical Biology, Medical University of Bialystok, 15-089 Białystok, Poland.ORCID 0000-0002-4901-5217

Funding

Medical University of Bialystok N/ST/MN/18/002/1117Medical University of Bialystok SUB/1/DN/19/003/1117
6 · The paper itself

Abstract

Adipose tissue (AT) is an endocrine organ involved in the management of energy metabolism via secretion of adipokines, hormones, and recently described secretory microvesicles, i.e., exosomes. Exosomes are rich in possible biologically active factors such as proteins, lipids, and RNA. The secretory function of adipose tissue is affected by pathological processes. One of the most important of these is obesity, which triggers adipose tissue inflammation and adversely affects the release of beneficial adipokines. Both processes may lead to further AT dysfunction, contributing to changes in whole-body metabolism and, subsequently, to insulin resistance. According to recent data, changes within the production, release, and content of exosomes produced by AT may be essential to understand the role of adipose tissue in the development of metabolic disorders. In this review, we summarize actual knowledge about the possible role of AT-derived exosomes in the development of insulin resistance, highlighting methodological challenges and potential gains resulting from exosome studies.

Indexed as

Insulin ResistanceAdipose TissueAnimalsExosomesGlucose IntoleranceHumansadipokinesadipose tissueexosomesinsulin resistancemetabolic disorderstype 2 diabetes

Identifiers

PMID34299048
PMCPMC8304687

What Socratic holds

Textmetadata
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.