Evidence mapPaperPMID 34299291Full record

ReviewInternational journal of molecular sciences2021

Hidden Role of Gut Microbiome Dysbiosis in Schizophrenia: Antipsychotics or Psychobiotics as Therapeutics?

Nayla Munawar, Khansa Ahsan, Khalid Muhammad, Aftab Ahmad, Munir A Anwar, Iltaf Shah, Ahlam Khalifa Al Ameri, Fadwa Al Mughairbi

Open access · goldAbstract readReview
In one paragraph

Review in International journal of molecular sciences, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 39 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
39citing papers in PubMed, 2 pooled it
5.4field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

39 citing papers in PubMed, 2 syntheses or guidelines pooled it, 96 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 3 institutions in 2 countries.

Nayla MunawarDepartment of Chemistry, College of Sciences, United Arabs Emirates University, Al Ain 15551, United Arab Emirates.
Khansa AhsanDepartment of Chemistry, College of Sciences, United Arabs Emirates University, Al Ain 15551, United Arab Emirates.
Khalid MuhammadDepartment of Biology, College of Sciences, United Arabs Emirates University, Al Ain 15551, United Arab Emirates.ORCID 0000-0001-6488-1722
Aftab AhmadDepartment of Biochemistry/Center for Advance Studies in Agriculture and Food Security (CAS-AFS), University of Agriculture, Faisalabad 38000, Pakistan.
Munir A AnwarIndustrial Biotechnology Division, National Institute for Biotechnology and Genetic Engineering, Constituent College of Pakistan Institute of Engineering and Applied Sciences, Faisalabad 38000, Pakistan.
Iltaf ShahDepartment of Chemistry, College of Sciences, United Arabs Emirates University, Al Ain 15551, United Arab Emirates.ORCID 0000-0002-3739-6083
Ahlam Khalifa Al AmeriDepartment of Chemistry, College of Sciences, United Arabs Emirates University, Al Ain 15551, United Arab Emirates.
Fadwa Al MughairbiDepartment of Psychiatry and Behavioral Science, United Arabs Emirates University, Al Ain 15551, United Arab Emirates.ORCID 0000-0002-6306-5632
United Arab Emirates University · AENational Institute for Biotechnology and Genetic Engineering · PKUniversity of Agriculture Faisalabad · PK

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Schizophrenia is a chronic, heterogeneous neurodevelopmental disorder that has complex symptoms and uncertain etiology. Mounting evidence indicates the involvement of genetics and epigenetic disturbances, alteration in gut microbiome, immune system abnormalities, and environmental influence in the disease, but a single root cause and mechanism involved has yet to be conclusively determined. Consequently, the identification of diagnostic markers and the development of psychotic drugs for the treatment of schizophrenia faces a high failure rate. This article surveys the etiology of schizophrenia with a particular focus on gut microbiota regulation and the microbial signaling system that correlates with the brain through the vagus nerve, enteric nervous system, immune system, and production of postbiotics. Gut microbially produced molecules may lay the groundwork for further investigations into the role of gut microbiota dysbiosis and the pathophysiology of schizophrenia. Current treatment of schizophrenia is limited to psychotherapy and antipsychotic drugs that have significant side effects. Therefore, alternative therapeutic options merit exploration. The use of psychobiotics alone or in combination with antipsychotics may promote the development of novel therapeutic strategies. In view of the individual gut microbiome structure and personalized response to antipsychotic drugs, a tailored and targeted manipulation of gut microbial diversity naturally by novel prebiotics (non-digestible fiber) may be a successful alternative therapeutic for the treatment of schizophrenia patients.

Indexed as

Antipsychotic AgentsBrainDysbiosisGastrointestinal MicrobiomeHumansImmune SystemPrebioticsProbioticsSchizophreniaAntipsychotic AgentsPrebioticsantipsychoticsdysbiosisgut microbiomepostbioticsprebioticsprobioticsschizophrenia

Identifiers

PMID34299291
PMCPMC8307070
OpenAlexW3184628058

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.