Evidence mapPaperPMID 34439436Full record

ArticleAntioxidants (Basel, Switzerland)2021

Noise-Induced Cochlear Damage Involves PPAR Down-Regulation through the Interplay between Oxidative Stress and Inflammation.

Fabiola Paciello, Anna Pisani, Rolando Rolesi, Vincent Escarrat, Jacopo Galli, Gaetano Paludetti, Claudio Grassi, Diana Troiani, Anna Rita Fetoni

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In one paragraph

Article in Antioxidants (Basel, Switzerland), 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

20 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Fabiola PacielloFondazione Policlinico Universitario A. Gemelli IRCCS, 00168 Roma, Italy.
Anna PisaniDepartment of Otolaryngology Head and Neck Surgery, Università Cattolica del Sacro Cuore, 00168 Roma, Italy.ORCID 0000-0003-0661-5017
Rolando RolesiDepartment of Otolaryngology Head and Neck Surgery, Università Cattolica del Sacro Cuore, 00168 Roma, Italy.
Vincent EscarratDepartment of Otolaryngology Head and Neck Surgery, Università Cattolica del Sacro Cuore, 00168 Roma, Italy.ORCID 0000-0002-4235-809X
Jacopo GalliFondazione Policlinico Universitario A. Gemelli IRCCS, 00168 Roma, Italy.
Gaetano PaludettiFondazione Policlinico Universitario A. Gemelli IRCCS, 00168 Roma, Italy.
Claudio GrassiFondazione Policlinico Universitario A. Gemelli IRCCS, 00168 Roma, Italy.ORCID 0000-0001-7253-1685
Diana TroianiDepartment of Neuroscience, Università Cattolica del Sacro Cuore, 00168 Roma, Italy.
Anna Rita FetoniFondazione Policlinico Universitario A. Gemelli IRCCS, 00168 Roma, Italy.

Funding

INAIL Bando BRIC 09
6 · The paper itself

Abstract

The cross-talk between oxidative stress and inflammation seems to play a key role in noise-induced hearing loss. Several studies have addressed the role of PPAR receptors in mediating antioxidant and anti-inflammatory effects and, although its protective activity has been demonstrated in several tissues, less is known about how PPARs could be involved in cochlear dysfunction induced by noise exposure. In this study, we used an in vivo model of noise-induced hearing loss to investigate how oxidative stress and inflammation participate in cochlear dysfunction through PPAR signaling pathways. Specifically, we found a progressive decrease in PPAR expression in the cochlea after acoustic trauma, paralleled by an increase in oxidative stress and inflammation. By comparing an antioxidant (Q-ter) and an anti-inflammatory (Anakinra) treatment, we demonstrated that oxidative stress is the primary element of damage in noise-induced cochlear injury and that increased inflammation can be considered a consequence of PPAR down-regulation induced by ROS production. Indeed, by decreasing oxidative stress, PPARs returned to control values, reactivating the negative control on inflammation in a feedback loop.

Indexed as

acoustic traumaAnakinracochleaIL-1βNF-κBQ-terROS

Identifiers

PMID34439436
PMCPMC8388985

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.