Evidence mapPaperPMID 34557159Full record

ArticleFrontiers in endocrinology2021

Differential Effects of Estrogen Receptor Alpha and Beta on Endogenous Ligands of Peroxisome Proliferator-Activated Receptor Gamma in Papillary Thyroid Cancer.

Shucai Yang, Zhongqin Gong, Zhimin Liu, Minghui Wei, Lingbin Xue, Alexander C Vlantis, Yang Zhang, Jason Yk Chan, C Andrew van Hasselt, Xianhai Zeng and 6 more

Open access · goldAbstract read
In one paragraph

Article in Frontiers in endocrinology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
1.1field-weighted citation impact, top 22% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 19 citations in OpenAlex.

  1. Review
  2. Article
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  5. The evolutionary impact and influence of oestrogens on adipose tissue structure and function.Philosophical transactions of the Royal Society of London. Series B, Biological sciences · 2023
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

16 authors at 7 institutions in 1 country.

Shucai YangDepartment of Clinical Laboratory, Pingshan District People's Hospital of Shenzhen, Shenzhen, China.
Zhongqin GongDepartment of Otorhinolaryngology, Head and Neck Surgery, The Chinese University of Hong Kong, Prince of Wales Hospital, Shatin, Hong Kong SAR, China.
Zhimin LiuDepartment of Biochemistry and Molecular Biology, Faculty of Basic Medical Sciences, Chongqing Medical University, Chongqing, China.
Minghui WeiDepartment of Head & Neck Surgery, Cancer Hospital Chinese Academy of Medical Sciences, Shenzhen Center, Shenzhen, China.
Lingbin XueDepartment of Otorhinolaryngology, Head and Neck Surgery, The Chinese University of Hong Kong, Prince of Wales Hospital, Shatin, Hong Kong SAR, China.
Alexander C VlantisDepartment of Otorhinolaryngology, Head and Neck Surgery, The Chinese University of Hong Kong, Prince of Wales Hospital, Shatin, Hong Kong SAR, China.
Yang ZhangDepartment of Clinical Laboratory, Pingshan District People's Hospital of Shenzhen, Shenzhen, China.
Jason Yk ChanDepartment of Otorhinolaryngology, Head and Neck Surgery, The Chinese University of Hong Kong, Prince of Wales Hospital, Shatin, Hong Kong SAR, China.
C Andrew van HasseltDepartment of Otorhinolaryngology, Head and Neck Surgery, The Chinese University of Hong Kong, Prince of Wales Hospital, Shatin, Hong Kong SAR, China.
Xianhai ZengShenzhen Key Laboratory of Ear, Nose and Throat (ENT), Institute of ENT & Longgang ENT Hospital, Shenzhen, China.
Shuqi QiuShenzhen Key Laboratory of Ear, Nose and Throat (ENT), Institute of ENT & Longgang ENT Hospital, Shenzhen, China.
Nelson TangDepartment of Chemical Pathology, The Chinese University of Hong Kong, Prince of Wales Hospital, Shatin, Hong Kong SAR, China.
Jing DuDepartment of Laboratory Medicine, Peking University Shenzhen Hospital, Shenzhen, China.
Wei WeiDepartment of Thyroid and Breast Surgery, Peking University Shenzhen Hospital, Shenzhen, China.
Michael Cf TongDepartment of Otorhinolaryngology, Head and Neck Surgery, The Chinese University of Hong Kong, Prince of Wales Hospital, Shatin, Hong Kong SAR, China.
George G ChenDepartment of Otorhinolaryngology, Head and Neck Surgery, The Chinese University of Hong Kong, Prince of Wales Hospital, Shatin, Hong Kong SAR, China.
Chinese University of Hong Kong · CNPrince of Wales Hospital · CNLonggang Central Hospital · CNPeking University Shenzhen Hospital · CNShenzhen Sixth People's Hospital · CNChinese Academy of Medical Sciences & Peking Union Medical College · CNChongqing Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Purpose: The inhibition of estrogen receptor alpha (ERα) or the activation of ERβ can inhibit papillary thyroid cancer (PTC), but the precise mechanism is not known. We aimed to explore the role of ERα and ERβ on the production of endogenous peroxisome proliferator-activated receptor gamma (PPARγ) ligands in PTC. Methods: 2 PTC cell lines, 32 pairs of PTC tissues and matched normal thyroid tissues were used in this study. The levels of endogenous PPARγ ligands 15(S)-hydroxyeicosatetraenoic acid (15(S)-HETE), 13-S-hydroxyoctadecadienoic acid (13(S)-HODE), and15-deoxy-Δ12,14-prostaglandin J2 (PGJ2) were measured by ELISA. Results: The levels of PGJ2 and 15(S)-HETE were significantly reduced in PTC, but 13(S)-HODE was not changed. Activation of ERα or inhibition of ERβ significantly downregulated the production of PGJ2, 15(S)-HETE and 13(S)-HODE, whereas inhibition of ERα or activation of ERβ markedly upregulated the production of these three ligands. Application of endogenous PPARγ ligands inhibited growth, induced apoptosis of cancer cells, and promoted the efficacy of chemotherapy. Conclusion: The levels of endogenous PPARγ ligands PGJ2 and 15(S)-HETE are significantly decreased in PTC. The inhibition of ERα or activation of ERβ can inhibit PTC by stimulating the production of endogenous PPARγ ligands to induce apoptosis in cancer cells.

Indexed as

AdultApoptosisCell MovementCell ProliferationEstrogen Receptor betaFemaleHumansHydroxyeicosatetraenoic AcidsLigandsLinoleic AcidsMaleMiddle AgedPPAR gammaPrognosisProstaglandin D2Thyroid Cancer, Papillary13-hydroxy-9,11-octadecadienoic acid15-hydroxy-5,8,11,13-eicosatetraenoic acid9-deoxy-delta-9-prostaglandin D2ESR2 protein, humanEstrogen Receptor betaHydroxyeicosatetraenoic AcidsLigandsLinoleic AcidsPPAR gammaProstaglandin D215(S)-HETEestrogen receptorspapillary thyroid cancerperoxisome proliferator-activated receptor gammaPGJ2

Identifiers

PMID34557159
PMCPMC8453163
OpenAlexW3197077477

What Socratic holds

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LicenceCC BY
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.