Evidence mapPaperPMID 34579756Full record

ArticleChinese medicine2021

Mechanism exploration of Gouqi-wentang formula against type 2 diabetes mellitus by phytochemistry and network pharmacology-based analysis and biological validation.

Lin Han, Hao-Yu Yang, Yu-Jiao Zheng, Xiu-Xiu Wei, Wen-Chao Dan, Li-Li Zhang, Qi-You Ding, Xu Ma, Xin-Miao Wang, Lin-Hua Zhao and 1 more

Open access · goldAbstract read
In one paragraph

Article in Chinese medicine, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
1.4field-weighted citation impact, top 20% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 9 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 4 institutions in 1 country.

Lin Han *Guang'anmen Hospital, China Academy of Chinese Medical Sciences, Beijing, 100053, China.
Hao-Yu Yang *Beijing University of Chinese Medicine, Beijing, 100029, China.
Yu-Jiao ZhengBeijing University of Chinese Medicine, Beijing, 100029, China.
Xiu-Xiu WeiBeijing University of Chinese Medicine, Beijing, 100029, China.
Wen-Chao DanBeijing University of Chinese Medicine, Beijing, 100029, China.
Li-Li ZhangGuang'anmen Hospital, China Academy of Chinese Medical Sciences, Beijing, 100053, China.
Qi-You DingBeijing University of Chinese Medicine, Beijing, 100029, China.
Xu MaGansu University of Chinese Medicine, Lanzhou, 730000, China.
Xin-Miao WangGuang'anmen Hospital, China Academy of Chinese Medical Sciences, Beijing, 100053, China.
Lin-Hua ZhaoGuang'anmen Hospital, China Academy of Chinese Medical Sciences, Beijing, 100053, China. melonzhao@163.com.
Xiao-Lin TongGuang'anmen Hospital, China Academy of Chinese Medical Sciences, Beijing, 100053, China. tongxiaolin@vip.163.com.
Beijing University of Chinese Medicine · CNGuang’anmen Hospital · CNChinese Academy of Medical Sciences & Peking Union Medical College · CNGansu University of Traditional Chinese Medicine · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundThe Gouqi-wentang formula (GQWTF) is a herbal formula used by Academician Xiao-lin Tong for the clinical treatment of T2DM. GQWTF is beneficial to qi, nourishes Yin, clears heat, and promotes fluid production, but the effective components and their mechanism of action remain unclear.

methodsThe main components of GQWTF were detected by LC-MS, and the multi-target mechanisms of GQWTF in T2DM were elucidated using network pharmacology analysis, including target prediction, protein-protein interaction network construction and analysis, Gene Ontology (GO) terms, Kyoto Encyclopaedia of Genes and Genomes (KEGG) pathway annotation, and other network construction. Finally, the efficacy of the GQWTF was verified using biological experiments.

resultsFirst, the "herb-channel tropism" network suggested that GQWTF focuses more on treating diseases by recuperating the liver, which is considered as an important insulin-sensitive organ. Subsequently, a total of 16 active ingredients in GQWTF were detected and screened, and their biological targets were predicted. Then, "compound-target" network was constructed, where enrichment analysis of GQWTF targets reflected its potential pharmacological activities. After T2DM-related target identification, 39 cross targets of GQWTF and T2DM were obtained, and 30 key targets highly responsible for the beneficial effect of GQWTF on T2DM were identified by PPI analysis. GO analysis of these key targets showed that many biological processes of GQWTF in treating T2DM are key in the occurrence and development of T2DM, including components related to inflammatory/immune response, insulin, and metabolism. KEGG analysis revealed the regulation of multiple signalling pathways, such as insulin resistance, PPAR signalling pathway, FoxO signalling pathway, Fc epsilon RI signalling pathway, and pathways that influence diabetes primarily by regulating metabolism as well as other T2DM directly related pathways. Furthermore, a "formula-compound-pathway-symptom" network was constructed to represent a global view of GQWTF in the treatment of T2DM.

conclusionsThis study explored the mechanism of action of GQWTF in T2DM by multi-component and multi-target multi pathways, which could provide a theoretical basis for the development and clinical application of GQWTF.

Indexed as

Biological validationGouqi-wentang formula (GQWTF)LC–MSNetwork pharmacologyT2DM

Identifiers

PMID34579756
PMCPMC8477540
OpenAlexW3204579577

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.