Evidence mapPaperPMID 34598978Full record

ArticleGut2022

Multiomics reveal unique signatures of human epiploic adipose tissue related to systemic insulin resistance.

Laura Krieg, Konrad Didt, Isabel Karkossa, Stephan H Bernhart, Stephanie Kehr, Narmadha Subramanian, Andreas Lindhorst, Alexander Schaudinn, Shirin Tabei, Maria Keller and 12 more

Open access · bronzeAbstract read
In one paragraph

Article in Gut, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 25 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
25citing papers in PubMed, 2 pooled it
3.7field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

25 citing papers in PubMed, 2 syntheses or guidelines pooled it, 38 citations in OpenAlex.

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  20. Adipose 'neighborhoods' collaborate to maintain metabolic health.Current opinion in genetics & development · 2023
    Review
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

22 authors at 9 institutions in 2 countries.

Laura Krieg *Department of Molecular Systems Biology, Helmholtz Centre for Environmental Research (UFZ), Leipzig, Germany.ORCID 0000-0002-1345-6651
Konrad Didt *Department for Internal Medicine, Neurology and Dermatology, University Hospital Leipzig, Leipzig, Germany.
Isabel KarkossaDepartment of Molecular Systems Biology, Helmholtz Centre for Environmental Research (UFZ), Leipzig, Germany.ORCID 0000-0003-2781-1877
Stephan H BernhartFaculty of Mathematics and Computer Science, Department of Computer Science and Interdisciplinary Center for Bioinformatics, University of Leipzig, Leipzig, Germany.
Stephanie KehrFaculty of Mathematics and Computer Science, Department of Computer Science and Interdisciplinary Center for Bioinformatics, University of Leipzig, Leipzig, Germany.ORCID 0000-0001-8461-5763
Narmadha SubramanianDepartment of Medicine (H7), Karolinska Institutet, Stockholm, Sweden.
Andreas LindhorstFaculty of Medicine, Institute of Anatomy, University of Leipzig, Leipzig, Germany.
Alexander SchaudinnDepartment of Diagnostic and Interventional Radiology, University Hospital Leipzig, Leipzig, Germany.
Shirin TabeiInstitute of Endocrinology and Diabetes, University of Lübeck, Lübeck, Germany.
Maria KellerHelmholtz Institute for Metabolic Obesity and Vascular Research (HI-MAG), Helmholtz Zentrum München, University of Leipzig and University Hospital Leipzig, Leipzig, Germany.
Michael StumvollMedical Department III - Endocrinology, Nephrology and Rheumatology, University of Leipzig Medical Center, Leipzig, Germany.
Arne DietrichClinic for Visceral, Transplantation and Thorax and Vascular Surgery, University Hospital Leipzig, Leipzig, Germany.
Martin von BergenDepartment of Molecular Systems Biology, Helmholtz Centre for Environmental Research (UFZ), Leipzig, Germany.ORCID 0000-0003-2732-2977
Peter F StadlerFaculty of Mathematics and Computer Science, Department of Computer Science and Interdisciplinary Center for Bioinformatics, University of Leipzig, Leipzig, Germany.ORCID 0000-0002-5016-5191
Jurga LaurencikieneDepartment of Medicine (H7), Karolinska Institutet, Stockholm, Sweden.
Martin KrügerFaculty of Medicine, Institute of Anatomy, University of Leipzig, Leipzig, Germany.
Matthias BlüherHelmholtz Institute for Metabolic Obesity and Vascular Research (HI-MAG), Helmholtz Zentrum München, University of Leipzig and University Hospital Leipzig, Leipzig, Germany.
Martin GerickeFaculty of Medicine, Institute of Anatomy, University of Leipzig, Leipzig, Germany.
Kristin SchubertDepartment of Molecular Systems Biology, Helmholtz Centre for Environmental Research (UFZ), Leipzig, Germany.ORCID 0000-0003-4365-084X
Peter KovacsMedical Department III - Endocrinology, Nephrology and Rheumatology, University of Leipzig Medical Center, Leipzig, Germany.ORCID 0000-0002-0290-5423
Rima ChakarounMedical Department III - Endocrinology, Nephrology and Rheumatology, University of Leipzig Medical Center, Leipzig, Germany rima.chakaroun@wlab.gu.se rima.chakaroun@medizin.uni-leipzig.de lucas.massier@ki.se.ORCID 0000-0001-9901-1815
Lucas MassierDepartment of Medicine (H7), Karolinska Institutet, Stockholm, Sweden rima.chakaroun@wlab.gu.se rima.chakaroun@medizin.uni-leipzig.de lucas.massier@ki.se.ORCID 0000-0002-6199-2643
Helmholtz Centre for Environmental Research · DELeipzig University · DEUniversity Hospital Leipzig · DEKarolinska Institutet · SEHelmholtz Zentrum München · DEGerman Center for Diabetes Research · DEMartin Luther University Halle-Wittenberg · DEMax Planck Institute for Mathematics in the Sciences · DEUniversity of Lübeck · DE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

objectiveHuman white adipose tissue (AT) is a metabolically active organ with distinct depot-specific functions. Despite their locations close to the gastrointestinal tract, mesenteric AT and epiploic AT (epiAT) have only scarcely been investigated. Here, we aim to characterise these ATs in-depth and estimate their contribution to alterations in whole-body metabolism.

designMesenteric, epiploic, omental and abdominal subcutaneous ATs were collected from 70 patients with obesity undergoing Roux-en-Y gastric bypass surgery. The metabolically well-characterised cohort included nine subjects with insulin sensitive (IS) obesity, whose AT samples were analysed in a multiomics approach, including methylome, transcriptome and proteome along with samples from subjects with insulin resistance (IR) matched for age, sex and body mass index (n=9). Findings implying differences between AT depots in these subgroups were validated in the entire cohort (n=70) by quantitative real-time PCR.

resultsWhile mesenteric AT exhibited signatures similar to those found in the omental depot, epiAT was distinct from all other studied fat depots. Multiomics allowed clear discrimination between the IS and IR states in all tissues. The highest discriminatory power between IS and IR was seen in epiAT, where profound differences in the regulation of developmental, metabolic and inflammatory pathways were observed. Gene expression levels of key molecules involved in AT function, metabolic homeostasis and inflammation revealed significant depot-specific differences with epiAT showing the highest expression levels.

conclusionMulti-omics epiAT signatures reflect systemic IR and obesity subphenotypes distinct from other fat depots. Our data suggest a previously unrecognised role of human epiploic fat in the context of obesity, impaired insulin sensitivity and related diseases.

Indexed as

Insulin ResistanceAdipose TissueHumansInsulinObesityProteomeInsulinProteomediabetes mellitusobesityobesity surgery

Identifiers

PMID34598978
PMCPMC9554031
OpenAlexW3202951828

What Socratic holds

Textmetadata
LicenceCC BY-NC
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.