Evidence mapPaperPMID 34703264Full record

ArticleClinical, cosmetic and investigational dermatology2021

Exploring the Protective and Reparative Mechanisms of

Xiuqin Shi, Wenjing Cheng, Qian Wang, Jiachan Zhang, Changtao Wang, Meng Li, Dan Zhao, Dongdong Wang, Quan An

Open access · goldAbstract read
In one paragraph

Article in Clinical, cosmetic and investigational dermatology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed, 1 pooled it
3.6field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 1 synthesis or guideline pooled it, 32 citations in OpenAlex.

  1. Pooled it
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  6. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 2 institutions in 1 country.

Xiuqin ShiChemistry and Materials Engineering, Beijing Technology and Business University, Beijing, 100048, People's Republic of China.
Wenjing ChengChemistry and Materials Engineering, Beijing Technology and Business University, Beijing, 100048, People's Republic of China.
Qian WangChemistry and Materials Engineering, Beijing Technology and Business University, Beijing, 100048, People's Republic of China.
Jiachan ZhangChemistry and Materials Engineering, Beijing Technology and Business University, Beijing, 100048, People's Republic of China.
Changtao WangChemistry and Materials Engineering, Beijing Technology and Business University, Beijing, 100048, People's Republic of China.
Meng LiChemistry and Materials Engineering, Beijing Technology and Business University, Beijing, 100048, People's Republic of China.
Dan ZhaoChemistry and Materials Engineering, Beijing Technology and Business University, Beijing, 100048, People's Republic of China.
Dongdong WangChemistry and Materials Engineering, Beijing Technology and Business University, Beijing, 100048, People's Republic of China.
Quan AnYunnan Baiyao Group Co., Ltd, Kunming, 650000, People's Republic of China.
Beijing Technology and Business University · CNYunnan Investment Group (China) · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

background

methodsSix polysaccharides were obtained from

resultsAll six polysaccharides showed the ability to scavenge free radicals and enhance the tolerance of human skin fibroblasts to H2O2 damage. Among them, GLP1 was selected and separated into two components (GLP1I and GLP1II). The results showed that GLP1, GLP1I and GLPII could significantly reduce the levels of reactive oxygen species (ROS) and malondialdehyde (MDA). The protective effect of GLP1II was stronger than that of positive control vitamin C. In addition, GLP1, GLP1I and GLP1II could significantly increase the levels of superoxide dismutase (SOD), catalase (CAT) and glutathione peroxidase (GSH-Px). And GLP1I works best in both ways. Meanwhile, Nrf2, a key regulator of keAP1-NRF2/ARE signaling pathway, was activated, while Keap1, a negative regulator, was inhibited, thus promoting the expression of downstream antioxidant enzyme genes (GSTs, GCLs, Nqo1, and Ho-1).

conclusionThe results showed that GLP could protect human skin fibroblasts from oxidative damage caused by H

Indexed as

Ganoderma lucidumKeap1-Nrf2/ARE signaling pathwayoxidative stresspolysaccharide

Identifiers

PMID34703264
PMCPMC8525518
OpenAlexW3206794751

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.