Evidence map›Paper›PMID 34713546›Full record

ReviewGenesis (New York, N.Y. : 2000)2021

Unraveling the complex genetics of neural tube defects: From biological models to human genomics and back.

Paul Wolujewicz, John W Steele, Julia A Kaltschmidt, Richard H Finnell, Margaret Elizabeth Ross

Open access · greenAbstract readReview
In one paragraph

Review in Genesis (New York, N.Y. : 2000), 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 24 papers.

0numbers the graph read from it
0cells of the map it votes in
24citing papers in PubMed
4.4field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

24 citing papers in PubMed, 40 citations in OpenAlex.

  1. Folic acid prevention of neural tube defects requires retinoic acid produced by ALDH1L1.Proceedings of the National Academy of Sciences of the United States of America · 2026
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  2. Article
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  4. Review
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  6. Whole exome-based variant profiling and functional network characterization in neural tube defects.Child's nervous system : ChNS : official journal of the International Society for Pediatric Neurosurgery · 2025
    Article
  7. Review
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  19. Implication of chromosomal microarray analysis prior to in-utero repair of fetal open neural tube defect.Ultrasound in obstetrics & gynecology : the official journal of the International Society of Ultrasound in Obstetrics and Gynecology · 2023
    Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 3 institutions in 1 country.

Paul WolujewiczCenter for Neurogenetics, Feil Family Brain & Mind Research Institute, Weill Cornell Medicine, New York, New York, USA.ORCID 0000-0003-2982-9448
John W SteeleCenter for Precision Environmental Health, Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas, USA.
Julia A KaltschmidtDepartment of Neurosurgery, Stanford University School of Medicine, Stanford, California, USA.
Richard H FinnellCenter for Precision Environmental Health, Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas, USA.
Margaret Elizabeth RossCenter for Neurogenetics, Feil Family Brain & Mind Research Institute, Weill Cornell Medicine, New York, New York, USA.ORCID 0000-0001-6440-8089
Baylor College of Medicine · USCornell University · USStanford University · US

Funding

Translational Research Support CoreP30ES030285 · NIEHS · BAYLOR COLLEGE OF MEDICINE · PI Fernanda Laezza, Cheryl L. Walker · 2019 to 2026
$14.7M
Risk Genes and Environment Interactions in NTDsP01HD067244 · NICHD · WEILL MEDICAL COLL OF CORNELL UNIV · PI ROSS, MARGARET ELIZABETH · 2011 to 2020
$12.6M
Molecular Mechanisms Regulating Inhibitory Circuitry in the Spinal CordR01NS083998 · NINDS · STANFORD UNIVERSITY · PI KALTSCHMIDT, JULIA ANNA · 2013 to 2023
$3.6M
Training Program in Developmental BiologyT32HD060600 · NICHD · WEILL MEDICAL COLL OF CORNELL UNIV · PI BAO, ZHIRONG, STUHLMANN, HEIDI · 2010 to 2024
$3.5M
Biomechanics of Neural Tube Development using Brillouin-OCT MultimodalityR01HD095520 · NICHD · UNIVERSITY OF HOUSTON · PI FINNELL, RICHARD H., LARIN, KIRILL V · 2018 to 2022
$3.3M
Role of Slc25a32 and Its Interaction with Lrp6 in the Etiology of Neural Tube DefectsR01HD100535 · NICHD · BAYLOR COLLEGE OF MEDICINE · PI FINNELL, RICHARD H., LEI, YUNPING · 2020 to 2024
$2.9M
MicroRNA regulation of neural tube closureR01HD098131 · NICHD · BAYLOR COLLEGE OF MEDICINE · PI FINNELL, RICHARD H., PARCHEM, RONALD J · 2020 to 2024
$2.8M
Training in Precision Environmental Health SciencesT32ES027801 · NIEHS · BAYLOR COLLEGE OF MEDICINE · PI Cheryl L. Walker · 2018 to 2026
$2.8M
The Role of GPR161 in the Etiology of Neural Tube DefectsR01HD093758 · NICHD · BAYLOR COLLEGE OF MEDICINE · PI FINNELL, RICHARD H., KIM, SUNG EUN · 2018 to 2022
$1.9M
NICHD NIH HHS P01 HD067244NICHD NIH HHS R01 HD093758NICHD NIH HHS R01 HD095520NICHD NIH HHS R01 HD098131NICHD NIH HHS R01 HD100535NICHD NIH HHS T32 HD060600NIEHS NIH HHS P30 ES030285NIEHS NIH HHS T32 ES027801NINDS NIH HHS R01 NS083998
6 · The paper itself

Abstract

Neural tube defects (NTDs) are a classic example of preventable birth defects for which there is a proven-effective intervention, folic acid (FA); however, further methods of prevention remain unrealized. In the decades following implementation of FA nutritional fortification programs throughout at least 87 nations, it has become apparent that not all NTDs can be prevented by FA. In the United States, FA fortification only reduced NTD rates by 28-35% (Williams et al., 2015). As such, it is imperative that further work is performed to understand the risk factors associated with NTDs and their underlying mechanisms so that alternative prevention strategies can be developed. However, this is complicated by the sheer number of genes associated with neural tube development, the heterogeneity of observable phenotypes in human cases, the rareness of the disease, and the myriad of environmental factors associated with NTD risk. Given the complex genetic architecture underlying NTD pathology and the way in which that architecture interacts dynamically with environmental factors, further prevention initiatives will undoubtedly require precision medicine strategies that utilize the power of human genomics and modern tools for assessing genetic risk factors. Herein, we review recent advances in genomic strategies for discovering genetic variants associated with these defects, and new ways in which biological models, such as mice and cell culture-derived organoids, are leveraged to assess mechanistic functionality, the way these variants interact with other genetic or environmental factors, and their ultimate contribution to human NTD risk.

Indexed as

AnimalsDisease Models, AnimalGenetic Predisposition to DiseaseGenomicsHumansMutationNeural Tube Defectsanencephalygenomicsin vitro modelsmouse modelsMyelomeningocelespina bifida

Identifiers

PMID34713546
PMCPMC8851409
OpenAlexW3208443599

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.