Evidence map›Paper›PMID 34779281›Full record

ReviewPhysiology (Bethesda, Md.)2022

Is It Good to Have a Stiff Aorta with Aging? Causes and Consequences.

Gary L Pierce, Thais A Coutinho, Lyndsey E DuBose, Anthony J Donato

Open access · greenAbstract readReview
In one paragraph

Review in Physiology (Bethesda, Md.), 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 40 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
40citing papers in PubMed, 1 pooled it
6.1field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

40 citing papers in PubMed, 1 synthesis or guideline pooled it, 53 citations in OpenAlex.

  1. Effects of excess sodium consumption on arterial function in C57BL/6 mice.American journal of physiology. Heart and circulatory physiology · 2024
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 4 institutions in 2 countries.

Gary L PierceDepartment of Health and Human Physiology, University of Iowa, Iowa City, Iowa.ORCID 0000-0001-7499-080X
Thais A CoutinhoDepartment of Medicine, University of Ottawa, Ottawa, Ontario, Canada.
Lyndsey E DuBoseDivision of Geriatrics, Department of Medicine, University of Colorado Anschutz Medical Campus, Aurora, Colorado.ORCID 0000-0002-3566-4531
Anthony J DonatoDepartment of Internal Medicine, University of Utah, Salt Lake City, Utah.
University of Colorado Anschutz Medical Campus · USUniversity of Iowa · USUniversity of Ottawa · CAUniversity of Utah · US

Funding

University of Colorado Aging Training GrantT32AG000279 · NIA · UNIVERSITY OF COLORADO DENVER · PI Kerrie Moreau · 2001 to 2026
$10.1M
IMPACT OF T CELLS ON AGE-RELATED VASCULAR DYSFUNCTION: A TRANSLATIONAL APPROACH - DIVERSITY SUPPLEMENTR01AG060395 · NIA · UNIVERSITY OF UTAH · PI DONATO, ANTHONY JOHN · 2019 to 2023
$2.5M
Sympathetic Regulation of Large Artery Stiffness in Humans with Age-Related Isolated Systolic HypertensionR01AG063790 · NIA · UNIVERSITY OF IOWA · PI PIERCE, GARY L. · 2020 to 2024
$1.7M
Telomere uncapping and arterial dysfunction: Novel mechanism and implications for agingR01AG050238 · NIA · UNIVERSITY OF UTAH · PI DONATO, ANTHONY JOHN · 2016 to 2020
$1.6M
Ovarian Hormone Regulation of Central and Cerebrovascular HemodynamicsF32AG071273 · NIA · UNIVERSITY OF COLORADO DENVER · PI DUBOSE, LYNDSEY · 2021 to 2022
$122k
NIA NIH HHS F32 AG071273NIA NIH HHS R01 AG050238NIA NIH HHS R01 AG060395NIA NIH HHS R01 AG063790NIA NIH HHS T32 AG000279
6 · The paper itself

Abstract

Aortic stiffness increases with advancing age, more than doubling during the human life span, and is a robust predictor of cardiovascular disease (CVD) clinical events independent of traditional risk factors. The aorta increases in diameter and length to accommodate growing body size and cardiac output in youth, but in middle and older age the aorta continues to remodel to a larger diameter, thinning the pool of permanent elastin fibers, increasing intramural wall stress and resulting in the transfer of load bearing onto stiffer collagen fibers. Whereas aortic stiffening in early middle age may be a compensatory mechanism to normalize intramural wall stress and therefore theoretically "good" early in the life span, the negative clinical consequences of accelerated aortic stiffening beyond middle age far outweigh any earlier physiological benefit. Indeed, aortic stiffness and the loss of the "windkessel effect" with advancing age result in elevated pulsatile pressure and flow in downstream microvasculature that is associated with subclinical damage to high-flow, low-resistance organs such as brain, kidney, retina, and heart. The mechanisms of aortic stiffness include alterations in extracellular matrix proteins (collagen deposition, elastin fragmentation), increased arterial tone (oxidative stress and inflammation-related reduced vasodilators and augmented vasoconstrictors; enhanced sympathetic activity), arterial calcification, vascular smooth muscle cell stiffness, and extracellular matrix glycosaminoglycans. Given the rapidly aging population of the United States, aortic stiffening will likely contribute to substantial CVD burden over the next 2-3 decades unless new therapeutic targets and interventions are identified to prevent the potential avalanche of clinical sequelae related to age-related aortic stiffness.

Indexed as

Cardiovascular DiseasesVascular StiffnessAdolescentAgedAgingAortaBlood PressureCollagenElastinHumansMiddle AgedCollagenElastinagingarterial stiffnessblood pressurecardiovascular diseasehypertensionpulse wave velocity

Identifiers

PMID34779281
PMCPMC8977146
OpenAlexW3212011084

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.