Evidence mapPaperPMID 34813630Full record

ArticlePloS one2021

Carotid plaque thickness is increased in chronic kidney disease and associated with carotid and coronary calcification.

Sasha S Bjergfelt, Ida M H Sørensen, Henrik Ø Hjortkjær, Nino Landler, Ellen L F Ballegaard, Tor Biering-Sørensen, Klaus F Kofoed, Theis Lange, Bo Feldt-Rasmussen, Henrik Sillesen and 2 more

Registry-linked trialAbstract read
In one paragraph

Article in PloS one, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It reports registered trial NCT05565976. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT05565976 phase2 / phase3unknown status

Dapagliflozin Effect in Cognitive Impairment in Stroke Trial

Ran2020Enrolled270Registered outcomes3Posted comparisons0ConditionsDementia, Vascular, Metabolic Syndrome, Mild Cognitive Impairment, Stroke, IschemicArmsAntidiabetic, Dapagliflozin 10mg Tab, Platelet Antiaggregant, Statins (Cardiovascular Agents)
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3 · Its place in the literature

Who cites it

12 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors.

Sasha S BjergfeltDepartment of Nephrology, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark.
Ida M H SørensenDepartment of Nephrology, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark.ORCID 0000-0003-1057-5812
Henrik Ø HjortkjærDepartment of Cardiology, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark.ORCID 0000-0001-5786-4561
Nino LandlerDepartment of Cardiology, Herlev-Gentofte Hospital, University of Copenhagen, Copenhagen, Denmark.ORCID 0000-0001-5255-787X
Ellen L F BallegaardDepartment of Nephrology, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark.
Tor Biering-SørensenDepartment of Cardiology, Herlev-Gentofte Hospital, University of Copenhagen, Copenhagen, Denmark.
Klaus F KofoedDepartment of Cardiology, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark.ORCID 0000-0001-9742-1554
Theis LangeDepartment of Public Health (Biostatistics), University of Copenhagen, Copenhagen, Denmark.
Bo Feldt-RasmussenDepartment of Nephrology, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark.
Henrik SillesenDepartment of Vascular Surgery, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark.
Christina ChristoffersenDepartment of Clinical Biochemistry, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark.
Susanne BroDepartment of Nephrology, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark.ORCID 0000-0003-3002-4188

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundChronic kidney disease accelerates both atherosclerosis and arterial calcification. The aim of the present study was to explore whether maximal carotid plaque thickness (cPTmax) was increased in patients with chronic kidney disease compared to controls and associated with cardiovascular disease and severity of calcification in the carotid and coronary arteries.

methodsThe study group consisted of 200 patients with chronic kidney disease stage 3 from the Copenhagen Chronic Kidney Disease Cohort and 121 age- and sex-matched controls. cPTmax was assessed by ultrasound and arterial calcification by computed tomography scanning.

resultsCarotid plaques were present in 58% of patients (n = 115) compared with 40% of controls (n = 48), p = 0.002. Among participants with plaques, cPTmax (median, interquartile range) was significantly higher in patients compared with controls (1.9 (1.4-2.3) versus 1.5 (1.2-1.8) mm), p = 0.001. Cardiovascular disease was present in 9% of patients without plaques (n = 85), 23% of patients with cPTmax 1.0-1.9 mm (n = 69) and 35% of patients with cPTmax >1.9 mm (n = 46), p = 0.001. Carotid and coronary calcium scores >400 were present in 0% and 4%, respectively, of patients with no carotid plaques, in 19% and 24% of patients with cPTmax 1.0-1.9 mm, and in 48% and 53% of patients with cPTmax >1.9 mm, p<0.001.

conclusionsThis is the first study showing that cPTmax is increased in patients with chronic kidney disease stage 3 compared to controls and closely associated with prevalent cardiovascular disease and severity of calcification in both the carotid and coronary arteries.

Indexed as

AgedCarotid ArteriesCarotid Artery DiseasesCoronary Artery DiseaseCoronary VesselsCross-Sectional StudiesFemaleHumansMaleMiddle AgedPlaque, AtheroscleroticRenal Insufficiency, ChronicVascular Calcification

Identifiers

PMID34813630
PMCPMC8610240

What Socratic holds

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Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.