Evidence map›Paper›PMID 34975883›Full record

ReviewFrontiers in immunology2021

The Role of Helper T Cells in Psoriasis.

Peng Hu, Mengyao Wang, Hu Gao, Ai Zheng, Jinhui Li, Dezhi Mu, Jiyu Tong

Registry-linked trialOpen access · goldAbstract readReview
In one paragraph

Review in Frontiers in immunology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT07352306 (The Efficacy of Topical Diacerein Cream in Patients With Psoriasis Vulgaris), which is not on this map. Cited by 104 papers, 4 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
104citing papers in PubMed, 4 pooled it
11.0field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT07352306 phase4not yet recruitingnot on this mapstarted 2026, after this paper: background citation

The Efficacy of Topical Diacerein Cream in Patients With Psoriasis Vulgaris

TypeinterventionalSponsorCairo UniversityRan2026 to 2027Enrolled60ConditionsPsoriasis VulgarisArmsDiacerein, Placebo
3 · Its place in the literature

Who cites it

104 citing papers in PubMed, 4 syntheses or guidelines pooled it, 187 citations in OpenAlex.

  1. Pooled it
  2. Pooled it
  3. Pooled it
  4. Pooled it
  5. Trial
  6. Trial
  7. Article
  8. Review
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  10. Article
  11. Article
  12. Article
  13. Review
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  15. Article
  16. The Role of Elafin in the Pathogenesis of Psoriasis.International journal of molecular sciences · 2026
    Article
  17. Review
  18. A review of core immuno-inflammatory mechanisms and key regulatory targets in psoriasis.American journal of clinical and experimental immunology · 2026
    Review
  19. Review
  20. Article

44 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 3 institutions in 1 country.

Peng HuWest China Second University Hospital, Sichuan University, Chengdu, China.
Mengyao WangWest China Second University Hospital, Sichuan University, Chengdu, China.
Hu GaoWest China Second University Hospital, Sichuan University, Chengdu, China.
Ai ZhengWest China Second University Hospital, Sichuan University, Chengdu, China.
Jinhui LiWest China Second University Hospital, Sichuan University, Chengdu, China.
Dezhi MuWest China Second University Hospital, Sichuan University, Chengdu, China.
Jiyu TongWest China Second University Hospital, Sichuan University, Chengdu, China.
West China Second University Hospital of Sichuan University · CNSichuan University · CNWest China Medical Center of Sichuan University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Psoriasis is a complex, chronic relapsing and inflammatory skin disorder with a prevalence of approximately 2% in the general population worldwide. Psoriasis can be triggered by infections, physical injury and certain drugs. The most common type of psoriasis is psoriasis vulgaris, which primarily features dry, well-demarcated, raised red lesions with adherent silvery scales on the skin and joints. Over the past few decades, scientific research has helped us reveal that innate and adaptive immune cells contribute to the chronic inflammatory pathological process of psoriasis. In particular, dysfunctional helper T cells (Th1, Th17, Th22, and Treg cells) are indispensable factors in psoriasis development. When stimulated by certain triggers, antigen-presenting cells (APCs) can release pro-inflammatory factors (IL-23, IFN-α and IL-12), which further activate naive T cells and polarize them into distinct helper T cell subsets that produce numerous cytokines, such as TNF, IFN-γ, IL-17 and IL-22, which act on keratinocytes to amplify psoriatic inflammation. In this review, we describe the function of helper T cells in psoriasis and summarize currently targeted anti-psoriatic therapies.

Indexed as

HumansInterleukin-22Interleukin-23InterleukinsJanus Kinase InhibitorsPsoriasisTh17 CellsTh1 CellsT-Lymphocytes, Helper-InducerT-Lymphocytes, RegulatoryInterleukin-22Interleukin-23InterleukinsJanus Kinase InhibitorsbiologicscytokinespsoriasisTh17Tregs

Identifiers

PMID34975883
PMCPMC8714744
OpenAlexW4200128258

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.