Evidence map›Paper›PMID 35164154›Full record

ArticleMolecules (Basel, Switzerland)2022

Guggulsterone Mediated JAK/STAT and PPAR-Gamma Modulation Prevents Neurobehavioral and Neurochemical Abnormalities in Propionic Acid-Induced Experimental Model of Autism.

Rishabh Khera, Sidharth Mehan, Sonalika Bhalla, Sumit Kumar, Abdulrahman Alshammari, Metab Alharbi, Satya Sai Sadhu

Abstract read
In one paragraph

Article in Molecules (Basel, Switzerland), 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 29 papers.

0numbers the graph read from it
0cells of the map it votes in
29citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

29 citing papers in PubMed.

  1. Autologous cord bloodWorld journal of clinical pediatrics · 2025
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  13. Plant-mediated green synthesis of gold nanoparticles using an aqueous extract ofSaudi pharmaceutical journal : SPJ : the official publication of the Saudi Pharmaceutical Society · 2024
    Article
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  15. Article
  16. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Rishabh KheraDepartment of Pharmacology, Neuropharmacology Division, ISF College of Pharmacy, Moga 142001, Punjab, India.
Sidharth MehanDepartment of Pharmacology, Neuropharmacology Division, ISF College of Pharmacy, Moga 142001, Punjab, India.ORCID 0000-0003-0034-835X
Sonalika BhallaDepartment of Pharmacology, Neuropharmacology Division, ISF College of Pharmacy, Moga 142001, Punjab, India.
Sumit KumarDepartment of Pharmacology, Neuropharmacology Division, ISF College of Pharmacy, Moga 142001, Punjab, India.
Abdulrahman AlshammariDepartment of Pharmacology and Toxicology, College of Pharmacy, King Saud University, P.O. Box 2455, Riyadh 11451, Saudi Arabia.
Metab AlharbiDepartment of Pharmacology and Toxicology, College of Pharmacy, King Saud University, P.O. Box 2455, Riyadh 11451, Saudi Arabia.
Satya Sai SadhuChemistry Department, Northern Michigan University, 1401, Presque, Isle, Marquette, MI 49855, USA.

Funding

Indo Soviet Friendship College of Pharmacy ISFCP/IAEC/CPCSEA/Meeting No: 27/2020/Protocol No.456
6 · The paper itself

Abstract

Autism spectrum disorder is a neurodevelopmental disorder marked by repetitive behaviour, challenges in verbal and non-verbal communication, poor socio-emotional health, and cognitive impairment. An increased level of signal transducer and activator of transcription 3 (STAT3) and a decreased level of peroxisome proliferator-activated receptor (PPAR) gamma have been linked to autism pathogenesis. Guggulsterone (GST) has a neuroprotective effect on autistic conditions by modulating these signalling pathways. Consequently, the primary objective of this study was to examine potential neuroprotective properties of GST by modulating JAK/STAT and PPAR-gamma levels in intracerebroventricular propionic acid (ICV PPA) induced experimental model of autism in adult rats. In this study, the first 11 days of ICV-PPA injections in rats resulted in autism-like behavioural, neurochemical, morphological, and histopathological changes. The above modifications were also observed in various biological samples, including brain homogenate, CSF, and blood plasma. GST was also observed to improve autism-like behavioural impairments in autistic rats treated with PPA, including locomotion, neuromuscular coordination, depression-like behaviour, spatial memory, cognition, and body weight. Prolonged GST treatment also restored neurochemical deficits in a dose-dependent manner. Chronic PPA administration increased STAT3 and decreased PPAR gamma in autistic rat brain, CSF, and blood plasma samples, which were reversed by GST. GST also restored the gross and histopathological alterations in PPA-treated rat brains. Our results indicate the neuroprotective effects of GST in preventing autism-related behavioural and neurochemical alterations.

Indexed as

AnimalsAutism Spectrum DisorderDisease Models, AnimalFemaleJanus KinasesMaleNeuroprotective AgentsPPAR gammaPregnenedionesPropionatesRatsRats, WistarSignal TransductionSTAT Transcription FactorsJanus KinasesNeuroprotective AgentsPPAR gammapregna-4,17-diene-3,16-dionePregnenedionesPropionatespropionic acidSTAT Transcription FactorsautismguggulsteroneJAK/STATneuroprotectionPPAR-gammapropionic acid

Identifiers

PMID35164154
PMCPMC8839522

What Socratic holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.