Evidence map›Paper›PMID 35328352›Full record

ArticleInternational journal of molecular sciences2022

Beneficial Effects of Dietary Nitrite on a Model of Nonalcoholic Steatohepatitis Induced by High-Fat/High-Cholesterol Diets in SHRSP5/Dmcr Rats: A Preliminary Study.

Kunihiro Sonoda, Yuka Kono, Kazuya Kitamori, Kazuo Ohtake, Sachiko Shiba, Keizo Kasono, Jun Kobayashi

Open access · goldAbstract read
In one paragraph

Article in International journal of molecular sciences, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.5field-weighted citation impact, top 38% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 3 citations in OpenAlex.

  1. Article
  2. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 1 country.

Kunihiro SonodaDepartment of Food and Nutritional Environment, College of Human Life and Environment, Kinjo Gakuin University, Nagoya 463-8521, Japan.
Yuka KonoDepartment of Food and Nutritional Environment, College of Human Life and Environment, Kinjo Gakuin University, Nagoya 463-8521, Japan.
Kazuya KitamoriDepartment of Food and Nutritional Environment, College of Human Life and Environment, Kinjo Gakuin University, Nagoya 463-8521, Japan.
Kazuo OhtakeDivision of Physiology, School of Pharmaceutical Sciences, Faculty of Pharmaceutical Science, Josai University, Saitama 350-0248, Japan.
Sachiko ShibaDivision of Physiology, School of Pharmaceutical Sciences, Faculty of Pharmaceutical Science, Josai University, Saitama 350-0248, Japan.
Keizo KasonoDivision of Physiology, School of Pharmaceutical Sciences, Faculty of Pharmaceutical Science, Josai University, Saitama 350-0248, Japan.
Jun KobayashiDivision of Pathophysiology, Department of Clinical Dietetics and Human Nutrition, Faculty of Pharmaceutical Science, Josai University, Saitama 350-0248, Japan.ORCID 0000-0002-3490-2980
Josai University · JPKinjo Gakuin University · JP

Funding

Japan Society for the Promotion of Science 20K19725Kinjo Gakuin University Research Grant 2019 Grant Research A
6 · The paper itself

Abstract

Nonalcoholic steatohepatitis (NASH) is a chronic liver disease that leads to liver cirrhosis and hepatocellular carcinoma. Endothelial dysfunction caused by hepatic lipotoxicity is an underlying NASH pathology observed in the liver and the cardiovascular system. Here, we evaluated the effect of dietary nitrite on a rat NASH model. Stroke-prone, spontaneously hypertensive 5/Dmcr rats were fed a high-fat/high-cholesterol diet to develop the NASH model, with nitrite or captopril (100 mg/L, each) supplementation in drinking water for 8 weeks. The effects of nitrite and captopril were evaluated using immunohistochemical analyses of the liver and heart tissues. Dietary nitrite suppressed liver fibrosis in the rats by reducing oxidative stress, as measured using the protein levels of nicotinamide adenine dinucleotide phosphate oxidase components and inflammatory cell accumulation in the liver. Nitrite lowered the blood pressure in hypertensive NASH rats and suppressed left ventricular chamber enlargement. Similar therapeutic effects were observed in a captopril-treated rat NASH model, suggesting the possibility of a common signaling pathway through which nitrite and captopril improve NASH pathology. In conclusion, dietary nitrite attenuates the development of NASH with cardiovascular involvement in rats and provides an alternative NASH therapeutic strategy.

Indexed as

Non-alcoholic Fatty Liver DiseaseAnimalsCaptoprilCholesterolDiet, High-FatDisease Models, AnimalLiverLiver CirrhosisNitritesRatsRats, Inbred SHRCaptoprilCholesterolNitritesangiotensin IIangiotensin II type 1 receptorcaptoprilendothelial dysfunctionnicotinamide adenine dinucleotide phosphate oxidasenitratenitric oxidenitritenonalcoholic fatty liver diseasenonalcoholic steatohepatitis

Identifiers

PMID35328352
PMCPMC8951310
OpenAlexW4221031039

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.