Evidence mapPaperPMID 35371044Full record

ReviewFrontiers in immunology2022

Role of Interleukin-17A in the Pathomechanisms of Periodontitis and Related Systemic Chronic Inflammatory Diseases.

Yi Feng, Zheng Chen, Shao-Qin Tu, Jia-Ming Wei, Yu-Luan Hou, Zhi-Li Kuang, Xiao-Ning Kang, Hong Ai

Open access · goldAbstract readReview
In one paragraph

Review in Frontiers in immunology, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 35 papers, 3 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
35citing papers in PubMed, 3 pooled it
7.1field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

35 citing papers in PubMed, 3 syntheses or guidelines pooled it, 49 citations in OpenAlex.

  1. Pooled it
  2. Pooled it
  3. Pooled it
  4. Observational
  5. Review
  6. Article
  7. Article
  8. Review
  9. Article
  10. Article
  11. Review
  12. Review
  13. Periodontitis Salivary Microbiota Exacerbates Murine Rheumatoid Arthritis via Gut Dysbiosis and Immune Dysregulation.FASEB journal : official publication of the Federation of American Societies for Experimental Biology · 2025
    Article
  14. Review
  15. The Role of IL-17 in Periodontitis and Its Systemic Connections.International journal of molecular sciences · 2025
    Review
  16. Review
  17. Anti-Inflammatory Effects ofCurrent issues in molecular biology · 2025
    Article
  18. Review
  19. Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 2 institutions in 1 country.

Yi FengDepartment of Stomatology, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Zheng ChenDepartment of Stomatology, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Shao-Qin TuDepartment of Stomatology, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Jia-Ming WeiDepartment of Stomatology, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Yu-Luan HouDepartment of Stomatology, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Zhi-Li KuangDepartment of Stomatology, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Xiao-Ning KangDepartment of Stomatology, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Hong AiDepartment of Stomatology, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China.
Sun Yat-sen University · CNThird Affiliated Hospital of Sun Yat-sen University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Periodontitis is a chronic inflammatory and destructive disease caused by periodontal microbial infection and mediated by host immune response. As the main cause of loosening and loss of teeth in adults, it is considered to be one of the most common and serious oral diseases in the world. The co-existence of periodontitis and systemic chronic inflammatory diseases such as rheumatoid arthritis, psoriasis, inflammatory bowel disease, diabetes and so on is very common. It has been found that interleukin-17A (IL-17A) secreted by various innate and adaptive immune cells can activate a series of inflammatory cascade reactions, which mediates the occurrence and development of periodontitis and related systemic chronic inflammatory diseases. In this work, we review the role of IL-17A in the pathomechanisms of periodontitis and related systemic chronic inflammatory diseases, and briefly discuss the therapeutic potential of cytokine targeted agents that modulate the IL-17A signaling. A deep understanding of the possible molecular mechanisms in the relationship between periodontitis and systemic diseases will help dentists and physicians update their clinical diagnosis and treatment ideas.

Indexed as

Arthritis, RheumatoidPeriodontitisPsoriasisChronic DiseaseCytokinesHumansInflammationInterleukin-17CytokinesIL17A protein, humanInterleukin-17chronic inflammatory diseasesIL-17Ainflammationperiodontitispsoriasisrheumatoid arthritis

Identifiers

PMID35371044
PMCPMC8968732
OpenAlexW4220877359

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.