Evidence mapPaperPMID 35380994Full record

ArticleJCI insight2022

Effects of elevation of ANP and its deficiency on cardiorenal function.

Daria V Ilatovskaya, Vladislav Levchenko, Kristen Winsor, Gregory R Blass, Denisha R Spires, Elizaveta Sarsenova, Iuliia Polina, Adrian Zietara, Mark Paterson, Alison J Kriegel and 1 more

Open access · goldAbstract read
In one paragraph

Article in JCI insight, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
3.4field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed, 21 citations in OpenAlex.

  1. Sex differences in the progression of salt-sensitive hypertension in aged Dahl salt-sensitive rats.American journal of physiology. Regulatory, integrative and comparative physiology · 2026
    Article
  2. Maresin 1 modulates renal and cardiac lipid profiles in hypertensive rats.American journal of physiology. Renal physiology · 2025
    Article
  3. Article
  4. Article
  5. Article
  6. The effects of the atrial natriuretic peptide deficiency on renal cortical mitochondrial bioenergetics in the Dahl SS rat.FASEB journal : official publication of the Federation of American Societies for Experimental Biology · 2024
    Article
  7. Article
  8. Article
  9. Role of Cardiac Natriuretic Peptides in Heart Structure and Function.International journal of molecular sciences · 2022
    Review
  10. Review
  11. Review
  12. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 3 institutions in 1 country.

Daria V IlatovskayaDepartment of Physiology, Medical College of Wisconsin, Milwaukee, Wisconsin, USA.
Vladislav LevchenkoDepartment of Physiology, Medical College of Wisconsin, Milwaukee, Wisconsin, USA.
Kristen WinsorDepartment of Physiology, Medical College of Wisconsin, Milwaukee, Wisconsin, USA.
Gregory R BlassDepartment of Physiology, Medical College of Wisconsin, Milwaukee, Wisconsin, USA.
Denisha R SpiresDepartment of Physiology, Medical College of Wisconsin, Milwaukee, Wisconsin, USA.
Elizaveta SarsenovaDepartment of Medicine, Division of Nephrology, Medical University of South Carolina, Charleston, South Carolina, USA.
Iuliia PolinaDepartment of Medicine, Division of Nephrology, Medical University of South Carolina, Charleston, South Carolina, USA.
Adrian ZietaraDepartment of Physiology, Medical College of Wisconsin, Milwaukee, Wisconsin, USA.
Mark PatersonDepartment of Physiology, Medical College of Wisconsin, Milwaukee, Wisconsin, USA.
Alison J KriegelDepartment of Physiology, Medical College of Wisconsin, Milwaukee, Wisconsin, USA.
Alexander StaruschenkoDepartment of Molecular Pharmacology and Physiology and.
Medical College of Wisconsin · USMedical University of South Carolina · USUniversity of South Florida · US

Funding

Transport and metabolism in the kidneyI01BX004024 · VA · JAMES A. HALEY VA MEDICAL CENTER · 2021 to 2025
BLRD VA I01 BX004024NHLBI NIH HHS P01 HL116264NHLBI NIH HHS R01 HL148114NHLBI NIH HHS R35 HL135749
6 · The paper itself

Abstract

Atrial natriuretic peptide (ANP), encoded by Nppa, is a vasodilatory hormone that promotes salt excretion. Genome-wide association studies identified Nppa as a causative factor of blood pressure development, and in humans, ANP levels were suggested as an indicator of salt sensitivity. This study aimed to provide insights into the effects of ANP on cardiorenal function in salt-sensitive hypertension. To address this question, hypertension was induced in SSNPPA-/- (KO of Nppa in the Dahl salt-sensitive [SS] rat background) or SSWT (WT Dahl SS) rats by a high-salt (HS) diet challenge (4% NaCl for 21 days). Chronic infusion of ANP in SSWT rats attenuated the increase in blood pressure and cardiorenal damage. Overall, the SSNPPA-/- strain demonstrated higher blood pressure and intensified cardiac fibrosis (with no changes in ejection fraction) compared with SSWT rats. Furthermore, SSNPPA-/- rats exhibited kidney hypertrophy and higher glomerular injury scores, reduced diuresis, and lower sodium and chloride excretion than SSWT when fed a HS diet. Additionally, the activity of epithelial Na+ channel (ENaC) was found to be increased in the collecting ducts of the SSNPPA-/- rats. Taken together, these data show promise for the therapeutic benefits of ANP and ANP-increasing drugs for treating salt-sensitive hypertension.

Indexed as

Atrial Natriuretic FactorHypertensionAnimalsBlood PressureGenome-Wide Association StudyRatsRats, Inbred DahlSodiumSodium Chloride, DietaryAtrial Natriuretic FactorSodiumSodium Chloride, DietaryHypertensionNephrologySodium channels

Identifiers

PMID35380994
PMCPMC9090260
OpenAlexW4226041855

What Socratic holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.