Evidence mapPaperPMID 35404040Full record

ReviewCurrent atherosclerosis reports2022

More than Just a Monolayer: the Multifaceted Role of Endothelial Cells in the Pathophysiology of Atherosclerosis.

Marion Mussbacher, Klaudia Schossleitner, Julia B Kral-Pointner, Manuel Salzmann, Astrid Schrammel, Johannes A Schmid

Open access · hybridAbstract readReview
In one paragraph

Review in Current atherosclerosis reports, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 44 papers.

0numbers the graph read from it
0cells of the map it votes in
44citing papers in PubMed
5.6field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

44 citing papers in PubMed, 71 citations in OpenAlex.

  1. Early atherogenesis: In search of etiology.International journal of cardiology. Cardiovascular risk and prevention · 2026
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 3 institutions in 1 country.

Marion MussbacherDepartment of Pharmacology and Toxicology, University of Graz, Graz, Austria. marion.mussbacher@uni-graz.at.ORCID 0000-0001-5346-6785
Klaudia SchossleitnerDepartment of Dermatology, Skin and Endothelium Research Division, Medical University of Vienna, Vienna, Austria.ORCID 0000-0003-1202-5961
Julia B Kral-PointnerLudwig Boltzmann Institute for Cardiovascular Research, Vienna, Austria.ORCID 0000-0002-9386-7855
Manuel SalzmannLudwig Boltzmann Institute for Cardiovascular Research, Vienna, Austria.
Astrid SchrammelDepartment of Pharmacology and Toxicology, University of Graz, Graz, Austria.
Johannes A SchmidInstitute of Vascular Biology and Thrombosis Research, Medical University Vienna, Schwarzspanierstr. 17, 1090, Vienna, Austria. johannes.schmid@meduniwien.ac.at.
Ludwig Boltzmann Institute for Cancer Research · ATMedical University of Vienna · ATUniversity of Graz · AT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

purpose of the reviewIn this review, we summarize current insights into the versatile roles of endothelial cells in atherogenesis. RECENT

findingsThe vascular endothelium represents the first barrier that prevents the entry of lipoproteins and leukocytes into the vessel wall, thereby controlling two key events in the pathogenesis of atherosclerosis. Disturbance of endothelial homeostasis increases vascular permeability, inflammation, and cellular trans-differentiation, which not only promotes the build-up of atherosclerotic plaques but is also involved in life-threatening thromboembolic complications such as plaque rupture and erosion. In this review, we focus on recent findings on endothelial lipoprotein transport, inflammation, cellular transitions, and barrier function. By using cutting-edge technologies such as single-cell sequencing, epigenetics, and cell fate mapping, novel regulatory mechanisms and endothelial cell phenotypes have been discovered, which have not only challenged established concepts of endothelial activation, but have also led to a different view of the disease.

Indexed as

AtherosclerosisPlaque, AtheroscleroticEndothelial CellsEndothelium, VascularHumansInflammationAtherosclerosisEndothelial cellsEndothelial dysfunction

Identifiers

PMID35404040
PMCPMC9162978
OpenAlexW4223428649

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.