Evidence mapPaperPMID 35478145Full record

ArticleIn vivo (Athens, Greece)

Association of Vitamin D Deficiency and Vitamin D Receptor Genetic Variants With Coronary Artery Disease in Type 2 Diabetic Saudi Patients.

Alaa Shafie, Ahmad El Askary, Mazen Almehmadi, Hatem H Allam, Lamiaa K Elsayyad, Asmaa F Hassan, Bader B Althobaiti, Amin Nadheef, Aisha H Alharthi, Amal F Gharib

Open access · diamondAbstract read
In one paragraph

Article in In vivo (Athens, Greece). The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
1.8field-weighted citation impact, top 15% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 12 citations in OpenAlex.

  1. Article
  2. Article
  3. Vitamin D Alleviates Type 2 Diabetes Mellitus by Mitigating Oxidative Stress-Induced Pancreatic β-Cell Impairment.Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association · 2023
    Article
  4. Article
  5. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 2 institutions in 1 country.

Alaa ShafieDepartment of Clinical Laboratory Sciences, College of Applied Medical Sciences, Taif University, Taif, Kingdom of Saudi Arabia.
Ahmad El AskaryDepartment of Clinical Laboratory Sciences, College of Applied Medical Sciences, Taif University, Taif, Kingdom of Saudi Arabia; a.elaskary@tu.edu.sa dr579@outlook.com.
Mazen AlmehmadiDepartment of Clinical Laboratory Sciences, College of Applied Medical Sciences, Taif University, Taif, Kingdom of Saudi Arabia.
Hatem H AllamDepartment of Physical Therapy, College of Applied Medical Sciences, Taif University, Taif, Kingdom of Saudi Arabia.
Lamiaa K ElsayyadDepartment of Physical Therapy, College of Applied Medical Sciences, Taif University, Taif, Kingdom of Saudi Arabia.
Asmaa F HassanDepartment of Medical Physiology, College of Medicine, Taif University, Taif, Kingdom of Saudi Arabia.
Bader B Althobaitidepartment of Clinical Laboratories, King Abdulaziz Specialist Hospital in Taif, Ministry of Health, Taif, Kingdom of Saudi Arabia.
Amin Nadheefdepartment of Clinical Laboratories, King Abdulaziz Specialist Hospital in Taif, Ministry of Health, Taif, Kingdom of Saudi Arabia.
Aisha H AlharthiDepartment of Clinical Laboratory Sciences, College of Applied Medical Sciences, Taif University, Taif, Kingdom of Saudi Arabia.
Amal F GharibDepartment of Clinical Laboratory Sciences, College of Applied Medical Sciences, Taif University, Taif, Kingdom of Saudi Arabia.
Taif University · SAKing Abdulaziz Hospital · SA

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

BACKGROUND/

aimVitamin D deficiency accelerates the onset of type 2 diabetes mellitus (T2DM). Polymorphisms in the vitamin D receptor (VDR) have been linked to coronary artery disease (CAD). This study aimed to evaluate the association of vitamin D deficiency and VDR polymorphism with CAD in T2DM. PATIENTS AND

methodsA total of 150 adult male and female subjects, aged from 40 to 60 years, were divided into three groups, each with 50 subjects; control group, T2DM, and T2DM with CAD. Fasting blood glucose (FBG), total cholesterol (TC), triglycerides (TG), HDL-C, LDL-C, glycosylated hemoglobin (HbA1c), and 25-hydroxyvitamin D (25-OH D) were assessed. VDR genotypes (BsmI, Taq1 and FOK1) were investigated by polymerase chain reaction fragment length polymorphism.

resultsThere was a significant negative correlation between serum 25-OH D and FBG, TC, TG, and LDL-C levels, and a positive correlation with HDL-C levels in all diabetic patient groups. The risk of CAD was markedly higher in the group of T2DM with CAD in comparison to the control (p<0.0001) and the T2DM group. Regarding Taq1, there was also a significantly higher risk of CAD in Tt+tt genotypes and t allele in the T2DM with CAD group compared to control (p<0.001, 0.031 respectively). In addition, 25-OH D concentrations and the prevalence of VDR polymorphisms (BsmI, Taq1) were correlated with the risk of CAD.

conclusionDeficiency of vitamin D and the prevalence of VDR polymorphisms (BsmI, Taq1) can serve as important markers for CAD.

Indexed as

Coronary Artery DiseaseDiabetes Mellitus, Type 2Vitamin D DeficiencyAdultCholesterol, LDLFemaleGenetic Predisposition to DiseaseHumansMaleReceptors, CalcitriolSaudi ArabiaCholesterol, LDLReceptors, CalcitriolVDR protein, human25-OH DCoronary artery diseasegene polymorphismSaudi patientsType 2 diabetes mellitus

Identifiers

PMID35478145
PMCPMC9087110
OpenAlexW4225102780

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.