Evidence map›Paper›PMID 35482137›Full record

ReviewReviews in endocrine & metabolic disorders2022

Food cue reactivity: Neurobiological and behavioral underpinnings.

Scott E Kanoski, Kerri N Boutelle

Open access · hybridAbstract readReview
In one paragraph

Review in Reviews in endocrine & metabolic disorders, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 42 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
42citing papers in PubMed, 2 pooled it
5.7field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

42 citing papers in PubMed, 2 syntheses or guidelines pooled it, 57 citations in OpenAlex.

  1. Pooled it
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  7. Nucleus of the solitary tract regulation of cue-induced appetitive behaviors via midbrain dopamine neurons.Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology · 2026
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  18. Combined torso and foot heat exposure reduces acuteTemperature (Austin, Tex.) · 2026
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 2 institutions in 1 country.

Scott E KanoskiDepartment of Biological Sciences, Human and Evolutionary Biology Section, University of Southern California, Los Angeles, CA, USA.ORCID 0000-0001-9526-206X
Kerri N BoutelleDepartment of Pediatrics, Herbert Wertheim School of Public Health and Human Longevity Science, and Psychiatry, University of California San Diego, San Diego, CA, USA. kboutelle@health.ucsd.edu.ORCID 0000-0002-9573-4765
University of California San Diego · USUniversity of Southern California · US

Funding

Higher-Order Neural Control of Food IntakeR01DK104897 · NIDDK · UNIVERSITY OF SOUTHERN CALIFORNIA · PI KANOSKI, SCOTT EDWARD · 2015 to 2024
$4.3M
Interactions between diet and cognitionR01DK123423 · NIDDK · UNIVERSITY OF SOUTHERN CALIFORNIA · PI Scott Edward Kanoski · 2019 to 2026
$3.4M
Targeting food cue responsiveness for weight lossR01DK122504 · NIDDK · UNIVERSITY OF CALIFORNIA, SAN DIEGO · PI BOUTELLE, KERRI N · 2020 to 2024
$3.4M
Control of feeding behavior by melanin-concentrating hormoneR01DK118402 · NIDDK · UNIVERSITY OF SOUTHERN CALIFORNIA · PI Scott Edward Kanoski · 2018 to 2026
$3.2M
Cue-Reward Learning and Weight Gain in YouthR01DK111106 · NIDDK · UNIVERSITY OF CALIFORNIA, SAN DIEGO · PI BOUTELLE, KERRI N · 2017 to 2021
$3.1M
Behavioral Phenotypes of Reward and Inhibition in Parent-Child Weight LossR01DK114794 · NIDDK · UNIVERSITY OF CALIFORNIA, SAN DIEGO · PI BOUTELLE, KERRI N · 2017 to 2021
$2.4M
NIDDK NIH HHS R01 DK104897NIDDK NIH HHS R01 DK111106NIDDK NIH HHS R01 DK114794NIDDK NIH HHS R01 DK118402NIDDK NIH HHS R01 DK122504NIDDK NIH HHS R01 DK123423NIH HHS DK104897NIH HHS DK111106NIH HHS DK114794NIH HHS DK123423
6 · The paper itself

Abstract

The modern obesogenic environment contains an abundance of food cues (e.g., sight, smell of food) as well cues that are associated with food through learning and memory processes. Food cue exposure can lead to food seeking and excessive consumption in otherwise food-sated individuals, and a high level of food cue responsivity is a risk factor for overweight and obesity. Similar food cue responses are observed in experimental rodent models, and these models are therefore useful for mechanistically identifying the neural circuits mediating food cue responsivity. This review draws from both experimental rodent models and human data to characterize the behavioral and biological processes through which food-associated stimuli contribute to overeating and weight gain. Two rodent models are emphasized - cue-potentiated feeding and Pavlovian-instrumental transfer - that provide insight in the neural circuits and peptide systems underlying food cue responsivity. Data from humans are highlighted that reveal physiological, psychological, and neural mechanisms that connect food cue responsivity with overeating and weight gain. The collective literature identifies connections between heightened food cue responsivity and obesity in both rodents and humans, and identifies underlying brain regions (nucleus accumbens, amygdala, orbitofrontal cortex, hippocampus) and endocrine systems (ghrelin) that regulate food cue responsivity in both species. These species similarities are encouraging for the possibility of mechanistic rodent model research and further human research leading to novel treatments for excessive food cue responsivity in humans.

Indexed as

CuesHyperphagiaFeeding BehaviorHumansObesityWeight GainConditioningFood cueObesityOvereatingPavlovian

Identifiers

PMID35482137
PMCPMC9307541
OpenAlexW4225091009

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.