Evidence mapPaperPMID 35511873Full record

ArticleThe Journal of clinical endocrinology and metabolism2022

Increased Adipose Tissue Indices of Androgen Catabolism and Aromatization in Women With Metabolic Dysfunction.

Giada Ostinelli, Sofia Laforest, Scott G Denham, Marie-Frederique Gauthier, Virginie Drolet-Labelle, Emma Scott, Frédéric-Simon Hould, Simon Marceau, Natalie Z M Homer, Catherine Bégin and 2 more

Open access · hybridAbstract read
In one paragraph

Article in The Journal of clinical endocrinology and metabolism, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 16 papers.

0numbers the graph read from it
0cells of the map it votes in
16citing papers in PubMed
2.7field-weighted citation impact, top 9% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

16 citing papers in PubMed, 24 citations in OpenAlex.

  1. Article
  2. Nr4a1 β-cell specific deletion impairs glucose tolerance in female mice.American journal of physiology. Endocrinology and metabolism · 2026
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  11. The Metabolic Syndrome, a Human Disease.International journal of molecular sciences · 2024
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 4 institutions in 2 countries.

Giada OstinelliCentre de recherche de l'Institut universitaire de cardiologie et pneumologie de Québec-Université Laval, Québec City, QC G1V 4G5, Canada.
Sofia LaforestCentre de recherche de l'Institut universitaire de cardiologie et pneumologie de Québec-Université Laval, Québec City, QC G1V 4G5, Canada.
Scott G DenhamMass Spectrometry Core, Edinburgh Clinical Research Facility, University/BHF, Cardiovascular Sciences, University of Edinburgh, Queen's Medical Research Institute, Edinburgh, EH16 4TJ, UK.
Marie-Frederique GauthierCentre de recherche de l'Institut universitaire de cardiologie et pneumologie de Québec-Université Laval, Québec City, QC G1V 4G5, Canada.
Virginie Drolet-LabelleÉcole de nutrition, Université Laval, Québec City, QC G1V 0A6, Canada.
Emma ScottFaculté de médecine, Université Laval, Québec City, QC G1V 0A6, Canada.
Frédéric-Simon HouldCentre de recherche de l'Institut universitaire de cardiologie et pneumologie de Québec-Université Laval, Québec City, QC G1V 4G5, Canada.
Simon MarceauCentre de recherche de l'Institut universitaire de cardiologie et pneumologie de Québec-Université Laval, Québec City, QC G1V 4G5, Canada.
Natalie Z M HomerMass Spectrometry Core, Edinburgh Clinical Research Facility, University/BHF, Cardiovascular Sciences, University of Edinburgh, Queen's Medical Research Institute, Edinburgh, EH16 4TJ, UK.
Catherine BéginCentre de recherche de l'Institut universitaire de cardiologie et pneumologie de Québec-Université Laval, Québec City, QC G1V 4G5, Canada.ORCID 0000-0003-0554-4181
Ruth AndrewMass Spectrometry Core, Edinburgh Clinical Research Facility, University/BHF, Cardiovascular Sciences, University of Edinburgh, Queen's Medical Research Institute, Edinburgh, EH16 4TJ, UK.ORCID 0000-0002-6916-2994
André TchernofCentre de recherche de l'Institut universitaire de cardiologie et pneumologie de Québec-Université Laval, Québec City, QC G1V 4G5, Canada.ORCID 0000-0002-2587-1000
Institut universitaire de cardiologie et de pneumologie de Québec · CAThe Queen's Medical Research Institute · GBUniversité Laval · CAUniversity of Strathclyde · GB

Funding

CIHR PJT-169083Foundation of the Quebec Heart and Lung Institute - Laval University
6 · The paper itself

Abstract

contextBody fat distribution is a risk factor for obesity-associated comorbidities, and adipose tissue dysfunction plays a role in this association. In humans, there is a sex difference in body fat distribution, and steroid hormones are known to regulate several cellular processes within adipose tissue.

objectiveOur aim was to investigate if intra-adipose steroid concentration and expression or activity of steroidogenic enzymes were associated with features of adipose tissue dysfunction in individuals with severe obesity.

methodsSamples from 40 bariatric candidates (31 women, 9 men) were included in the study. Visceral (VAT) and subcutaneous adipose tissue (SAT) were collected during surgery. Adipose tissue morphology was measured by a combination of histological staining and semi-automated quantification. Following extraction, intra-adipose and plasma steroid concentrations were determined by liquid chromatography electrospray ionization tandem mass spectrometry (LC-ESI-MS/MS). Aromatase activity was estimated using product over substrate ratio, while AKR1C2 activity was measured directly by fluorogenic probe. Gene expression was measured by quantitative PCR.

resultsVAT aromatase activity was positively associated with VAT adipocyte hypertrophy (P valueadj < 0.01) and negatively with plasma high-density lipoprotein (HDL)-cholesterol (P valueadj < 0.01), while SAT aromatase activity predicted dyslipidemia in women even after adjustment for waist circumference, age, and hormonal contraceptive use. We additionally compared women with high and low visceral adiposity index (VAI) and found that VAT excess is characterized by adipose tissue dysfunction, increased androgen catabolism mirrored by increased AKR1C2 activity, and higher aromatase expression and activity indices.

conclusionIn women, increased androgen catabolism or aromatization is associated with visceral adiposity and adipose tissue dysfunction.

Indexed as

Adipose TissueAndrogensAromataseObesity, MorbidBody Fat DistributionBody Mass IndexFemaleGonadal Steroid HormonesHumansIntra-Abdominal FatMaleTandem Mass SpectrometryAndrogensAromataseGonadal Steroid Hormonesadipose tissueAKR1C2androgensaromataseobesitysex steroids

Identifiers

PMID35511873
PMCPMC9282357
OpenAlexW4229033140

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.