Evidence map›Paper›PMID 35546649›Full record

ArticleCardiovascular toxicology2022

Mesenchymal Stem Cell-Derived Exosome-Loaded microRNA-129-5p Inhibits TRAF3 Expression to Alleviate Apoptosis and Oxidative Stress in Heart Failure.

Fang Yan, Wei Cui, Ziying Chen

Abstract read
PubMed Publisher
In one paragraph

Article in Cardiovascular toxicology, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 32 papers.

0numbers the graph read from it
0cells of the map it votes in
32citing papers in PubMed
3.8field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

32 citing papers in PubMed, 47 citations in OpenAlex.

  1. Review
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  3. Exosomal miRNAs: A New Frontier in Cardiovascular Disease Diagnosis and Treatment.Journal of cardiovascular translational research · 2025
    Review
  4. Exploring the landscape of exosomes in heart failure: a bibliometric analysis.International journal of surgery (London, England) · 2025
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  14. The Potential of Mesenchymal Stem Cell-Derived Exosomes in Cardiac Repair.International journal of molecular sciences · 2024
    Review
  15. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Fang YanHebei Medical University, Shijiazhuang, 050000, Hebei, People's Republic of China.
Wei CuiDepartment of Cardiology, the Second Hospital of Hebei Medical University, No. 215, Heping West Road, Xinhua District, Shijiazhuang, 050000, Hebei, People's Republic of China. cuiwei6501@163.com.
Ziying ChenDepartment of Cardiac Surgery, The Second Hospital of Hebei Medical University, No. 215, Heping West Road, Xinhua District, Shijiazhuang, 050000, Hebei, People's Republic of China. hbchenzy64@126.com.ORCID 0000-0002-6627-5176
Hebei Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Heart failure (HF) represents a main global healthy and economic burden with unacceptably high morbidity and mortality rates. In the current study, we evaluated the potential effect of mesenchymal stem cell (MSC)-derived exosomes (MSC-Exos) on oxygen-glucose deprivation (OGD)-induced damages to HL-1 cells and HF mice and searched for the possible mechanism. MSC-Exos ameliorated oxidative stress and reduced apoptosis in OGD-treated HL-1 cells. By microarray analysis, we found that MSC-Exos treatment significantly increased the microRNA (miR)-129-5p expression in HL-1 cells. miR-129-5p inhibitor attenuated the protective effect of MSC-Exos on OGD-treated HL-1 cells. miR-129-5p targeted tumor necrosis factor receptor-associated factor 3 (TRAF3), and TRAF3 loss reversed the effect of miR-129-5p inhibitor by blunting the NF-κB signaling. MSC-Exos injection alleviated ventricular dysfunction and suppressed oxidative stress, apoptosis, inflammation, and fibrosis in cardiomyocytes in mice with HF by inhibiting NF-κB signaling pathway through miR-129-5p/TRAF3. Our findings suggest that exosomal miR-129-5p from MSCs protects the heart from failure by targeting TRAF3 and the following NF-κB signaling. This regulatory axis may be a possible therapeutic target for HF.

Indexed as

ExosomesHeart FailureMesenchymal Stem CellsAnimalsApoptosisHumansMiceMicroRNAsNF-kappa BOxidative StressTNF Receptor-Associated Factor 3MicroRNAsMirn129 microRNA, humanMIRN129 microRNA, mouseNF-kappa BTNF Receptor-Associated Factor 3TRAF3 protein, humanMesenchymal stem cells-derived exosomesmicroRNA-129-5pNF-κB signalingOxidative stressTRAF3

Identifiers

PMID35546649
OpenAlexW4280600785

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.