Evidence mapPaperPMID 35549375Full record

ReviewCirculation research2022

Cardiorenal Syndrome: The Role of Neural Connections Between the Heart and the Kidneys.

Kaushik P Patel, Kenichi Katsurada, Hong Zheng

Open access · bronzeAbstract readReview
In one paragraph

Review in Circulation research, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 53 papers.

0numbers the graph read from it
0cells of the map it votes in
53citing papers in PubMed
10.0field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

53 citing papers in PubMed, 70 citations in OpenAlex.

  1. Review
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  9. Cardiac and renal sympathetic nerve activation in early-stage HFpEF: insights fromThe international journal of cardiovascular imaging · 2026
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  10. Review
  11. Application ofAmerican journal of translational research · 2026
    Article
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  13. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 3 institutions in 2 countries.

Kaushik P PatelDepartment of Cellular and Integrative Physiology, University of Nebraska Medical Center, Omaha (K.P.P.).ORCID 0000-0002-4500-0819
Kenichi KatsuradaDivision of Cardiovascular Medicine, Department of Internal Medicine (K.K.), Jichi Medical University School of Medicine, Shimotsuke, Tochigi, Japan.ORCID 0000-0002-0567-1458
Hong ZhengBasic Biomedical Sciences, Sanford School of Medicine, University of South Dakota, Vermillion (H.Z.).ORCID 0000-0002-1660-3573
Jichi Medical University · JPUniversity of Nebraska Medical Center · USUniversity of South Dakota · US

Funding

SYMPATHETIC OUTFLOW IN HEART FAILURE--ANGIOTENSIN II AND NITRIC OXIDEP01HL062222 · UNIVERSITY OF NEBRASKA MEDICAL CENTER · 1999 to 2005
$8.6M
NHLBI NIH HHS P01 HL062222NHLBI NIH HHS R56 HL124104NIDDK NIH HHS R01 DK114663NIDDK NIH HHS R01 DK129311
6 · The paper itself

Abstract

The maintenance of cardiovascular homeostasis is highly dependent on tightly controlled interactions between the heart and the kidneys. Therefore, it is not surprising that a dysfunction in one organ affects the other. This interlinking relationship is aptly demonstrated in the cardiorenal syndrome. The characteristics of the cardiorenal syndrome state include alterations in neurohumoral drive, autonomic reflexes, and fluid balance. The evidence suggests that several factors contribute to these alterations. These may include peripheral and central nervous system abnormalities. However, accumulating evidence from animals with experimental models of congestive heart failure and renal dysfunction as well as humans with the cardiorenal syndrome suggests that alterations in neural pathways, from and to the kidneys and the heart, including the central nervous system are involved in regulating sympathetic outflow and may be critically important in the alterations in neurohumoral drive, autonomic reflexes, and fluid balance commonly observed in the cardiorenal syndrome. This review focuses on studies implicating neural pathways, particularly the afferent and efferent signals from the heart and the kidneys integrating at the level of the paraventricular nucleus in the hypothalamus to alter neurohumoral drive, autonomic pathways, and fluid balance. Further, it explores the potential mechanisms of action for the known beneficial use of various medications or potential novel therapeutic manipulations for the treatment of the cardiorenal syndrome. A comprehensive understanding of these mechanisms will enhance our ability to treat cardiorenal conditions and their cardiovascular complications more efficaciously and thoroughly.

Indexed as

Cardio-Renal SyndromeHeart FailureAnimalsFemaleHeartHumansKidneyMaleParaventricular Hypothalamic NucleusSympathetic Nervous Systemheart failurehomeostasishumanskidneyparaventricular hypothalamic nucleus

Identifiers

PMID35549375
PMCPMC9179008
OpenAlexW4280541414

What Socratic holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.