Evidence mapPaperPMID 35563296Full record

ReviewInternational journal of molecular sciences2022

Macrophage Migration Inhibitory Factor (MIF) as a Stress Molecule in Renal Inflammation.

Yao-Zhong Kong, Qiyan Chen, Hui-Yao Lan

Open access · goldAbstract readReview
In one paragraph

Review in International journal of molecular sciences, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 28 papers.

0numbers the graph read from it
0cells of the map it votes in
28citing papers in PubMed
3.2field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

28 citing papers in PubMed, 39 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Article
  6. Recombinant Macrophage Migration Inhibitory Factor Derived fromInternational journal of molecular sciences · 2026
    Article
  7. Review
  8. Identification of a novel and high affinity MIF inhibitorJournal of enzyme inhibition and medicinal chemistry · 2025
    Article
  9. Article
  10. Article
  11. Article
  12. Article
  13. Article
  14. Review
  15. Article
  16. Article
  17. Article
  18. Targeting Macrophages: Therapeutic Approaches in Diabetic Kidney Disease.International journal of molecular sciences · 2024
    Review
  19. Review
  20. Impact of theFrontiers in cardiovascular medicine · 2024
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 2 countries.

Yao-Zhong KongNephrology Department, The First People's Hospital of Foshan, Foshan 528000, China.
Qiyan ChenNephrology Department, The First People's Hospital of Foshan, Foshan 528000, China.
Hui-Yao LanDepartment of Medicine & Therapeutics, Li Ka Shing Institute of Health Sciences, The Chinese University of Hong Kong, Hong Kong SAR, China.ORCID 0000-0003-4283-9755
First People's Hospital of Foshan · CNChinese University of Hong Kong · HK

Funding

Deng Feng Program of Foshan 2019A007, 2020A017Lui Che Woo Institute of Innovative Medicine (CARE program)National Natural Science Foundation of China Grant 82100736Postdoctoral Science Foundation of China grant 2021M700778Research Grants of Council of Hong Kong 14117418, 14104019, 14101121
6 · The paper itself

Abstract

Renal inflammation is an initial pathological process during progressive renal injury regardless of the initial cause. Macrophage migration inhibitory factor (MIF) is a truly proinflammatory stress mediator that is highly expressed in a variety of both inflammatory cells and intrinsic kidney cells. MIF is released from the diseased kidney immediately upon stimulation to trigger renal inflammation by activating macrophages and T cells, and promoting the production of proinflammatory cytokines, chemokines, and stress molecules via signaling pathways involving the CD74/CD44 and chemokine receptors CXCR2, CXCR4, and CXCR7 signaling. In addition, MIF can function as a stress molecule to counter-regulate the immunosuppressive effect of glucocorticoid in renal inflammation. Given the critical position of MIF in the upstream inflammatory cascade, this review focuses on the regulatory role and molecular mechanisms of MIF in kidney diseases. The therapeutic potential of targeting MIF signaling to treat kidney diseases is also discussed.

Indexed as

Macrophage Migration-Inhibitory FactorsNephritisFemaleHistocompatibility Antigens Class IIHumansInflammationIntramolecular OxidoreductasesMaleReceptors, Interleukin-8BSignal TransductionStress, PhysiologicalHistocompatibility Antigens Class IIIntramolecular OxidoreductasesMacrophage Migration-Inhibitory FactorsMIF protein, humanReceptors, Interleukin-8Binflammationkidney diseasesmacrophagesMIFT cells

Identifiers

PMID35563296
PMCPMC9102975
OpenAlexW4224991039

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.