Evidence map›Paper›PMID 35565348›Full record

ReviewCancers2022

Tumor-Associated Macrophages and Ovarian Cancer: Implications for Therapy.

David Schweer, Annabel McAtee, Khaga Neupane, Christopher Richards, Frederick Ueland, Jill Kolesar

Abstract readReview
In one paragraph

Review in Cancers, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 91 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
91citing papers in PubMed, 2 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

91 citing papers in PubMed, 2 syntheses or guidelines pooled it.

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31 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

David SchweerMarkey Cancer Center, Division of Gynecologic Oncology, University of Kentucky, Lexington, KY 40536, USA.ORCID 0000-0001-7706-5206
Annabel McAteeSchool of Medicine, University of Kentucky, Lexington, KY 40536, USA.
Khaga NeupaneDepartment of Chemistry, College of Arts and Sciences, University of Kentucky, Lexington, KY 40536, USA.
Christopher RichardsDepartment of Chemistry, College of Arts and Sciences, University of Kentucky, Lexington, KY 40536, USA.
Frederick UelandMarkey Cancer Center, Division of Gynecologic Oncology, University of Kentucky, Lexington, KY 40536, USA.ORCID 0000-0003-1213-7509
Jill KolesarDepartment of Pharmacology and Toxicology, University of Kentucky, Lexington, KY 40202, USA.ORCID 0000-0003-1802-6439

Funding

University of Kentucky Markey Cancer Center Support Grant ECIA SupplementP30CA177558 · NCI · UNIVERSITY OF KENTUCKY · PI Bernard Mark Evers · 2013 to 2026
$38.3M
Oncology Research Training for Surgeon-ScientistsT32CA160003 · NCI · UNIVERSITY OF KENTUCKY · PI Bernard Mark Evers, Pamela Carmen Hull · 2011 to 2026
$3.2M
NCI NIH HHS P30 CA177558NCI NIH HHS T32 CA160003NIH HHS T32CA160003
6 · The paper itself

Abstract

The tumor microenvironment (TME) has been implicated to play an important role in the progression of ovarian cancer. One of the most important components of the TME is tumor associated macrophages (TAMs). Phenotypically, macrophages are broadly categorized as M1 pro-inflammatory or M2 anti-inflammatory, based on the cytokines and chemokines that they secrete. The tumor microenvironment is associated with macrophages of an M2 phenotype which suppress the surrounding immune environment, assist tumor cells in evading immune targeting, and support tumor growth and metastasis. Contrarily, M1 macrophages help mount an immune response against tumors, and are associated with a more favorable prognosis in solid tumors. One of the characteristic indicators of a poor prognosis in ovarian cancer is the overrepresentation of M2-type TAMs. As such, therapeutic modalities targeting TME and TAMs are of increasing interest. Pharmacological approaches to eliminate TAMs, include decreasing macrophage survival and recruitment and increasing phagocytosis, have been underwhelming. Clinical strategies targeting these macrophage subtypes via repolarization to an M1 antitumoral state deserve increasing attention, and may serve as a new modality for immunotherapy.

Indexed as

immunotherapymacrophagesovarian cancerrepolarizationTAM

Identifiers

PMID35565348
PMCPMC9101750

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.