Evidence map›Paper›PMID 35784371›Full record

ArticleFrontiers in immunology2022

UVB-Induced Skin Autoinflammation Due to

Yuya Murase, Takuya Takeichi, Jun Koseki, Yuki Miyasaka, Yoshinao Muro, Tamio Ohno, Teppei Shimamura, Masashi Akiyama

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Frontiers in immunology, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.5field-weighted citation impact, top 39% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 7 citations in OpenAlex.

  1. Research progress in the role of ultraviolet in the pathogenesis of rosacea.Zhong nan da xue xue bao. Yi xue ban = Journal of Central South University. Medical sciences · 2025
    Review
  2. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 1 institution in 1 country.

Yuya MuraseDepartment of Dermatology, Nagoya University Graduate School of Medicine, Nagoya, Japan.
Takuya TakeichiDepartment of Dermatology, Nagoya University Graduate School of Medicine, Nagoya, Japan.
Jun KosekiDivision of Systems Biology, Nagoya University Graduate School of Medicine, Nagoya, Japan.
Yuki MiyasakaDivision of Experimental Animals, Nagoya University Graduate School of Medicine, Nagoya, Japan.
Yoshinao MuroDepartment of Dermatology, Nagoya University Graduate School of Medicine, Nagoya, Japan.
Tamio OhnoDivision of Experimental Animals, Nagoya University Graduate School of Medicine, Nagoya, Japan.
Teppei ShimamuraDivision of Systems Biology, Nagoya University Graduate School of Medicine, Nagoya, Japan.
Masashi AkiyamaDepartment of Dermatology, Nagoya University Graduate School of Medicine, Nagoya, Japan.
Nagoya University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

NLRP1 (NACHT and leucine-rich repeat-containing protein family, pyrin domain-containing protein 1) is an innate immune sensor that is involved in the formation of inflammasome complexes. NLRP1 hyperactivity has been reported to cause inherited autoinflammatory diseases including familial keratosis lichenoides chronica and NLRP1-associated autoinflammation with arthritis and dyskeratosis. We generated

Indexed as

InflammasomesSkin DiseasesAnimalsApoptosis Regulatory ProteinsCaspase 1InflammationInterleukin-1betaMiceMutationUltraviolet RaysApoptosis Regulatory ProteinsCaspase 1IL1B protein, mouseInflammasomesInterleukin-1betaNalp1b protein, mouseautoinflammationIL-18IL-1βinflammasomeNLRP1NLRP1Bskin - immunologyUVB

Identifiers

PMID35784371
PMCPMC9248282
OpenAlexW4283073448

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.