Evidence mapPaperPMID 35806367Full record

ReviewInternational journal of molecular sciences2022

Phosphate in the Context of Cognitive Impairment and Other Neurological Disorders Occurrence in Chronic Kidney Disease.

Merita Rroji, Andreja Figurek, Davide Viggiano, Giovambattista Capasso, Goce Spasovski

Open access · goldAbstract readReview
In one paragraph

Review in International journal of molecular sciences, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
3.9field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed, 31 citations in OpenAlex.

  1. Observational
  2. Review
  3. Article
  4. Observational
  5. Article
  6. Article
  7. Article
  8. Soft tissue calcifications in chronic kidney disease-beyond the vasculature.Pflugers Archiv : European journal of physiology · 2025
    Review
  9. Review
  10. Article
  11. Genetic and circulating biomarkers of cognitive dysfunction and dementia in CKD.Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association · 2025
    Review
  12. Article
  13. Article
  14. Review
  15. Article
  16. Article
  17. Secondary Hyperparathyroidism and Cognitive Decline.Current transplantation reports · 2023
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 4 institutions in 5 countries.

Merita RrojiDepartment of Nephrology, Faculty of Medicine, University of Medicine Tirana, 1001 Tirana, Albania.ORCID 0000-0003-1048-2579
Andreja FigurekDepartment of Internal Medicine, Medical Faculty, University of Banja Luka, 78000 Banja Luka, Bosnia and Herzegovina.ORCID 0000-0003-3766-0312
Davide ViggianoDepartment of Translational Medical Sciences, University of Campania "L. Vanvitelli", 80138 Naples, Italy.ORCID 0000-0002-2425-6057
Giovambattista CapassoDepartment of Translational Medical Sciences, University of Campania "L. Vanvitelli", 80138 Naples, Italy.
Goce SpasovskiUniversity Clinic for Nephrology, Medical Faculty, University St. Cyril and Methodius, 1000 Skopje, North Macedonia.
University of Campania "Luigi Vanvitelli" · ITSs. Cyril and Methodius University in Skopje · MKUniversity of Banja Luka · BAUniversity of Medicine Tirana · AL

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The nervous system and the kidneys are linked under physiological states to maintain normal body homeostasis. In chronic kidney disease (CKD), damaged kidneys can impair the central nervous system, including cerebrovascular disease and cognitive impairment (CI). Recently, kidney disease has been proposed as a new modifiable risk factor for dementia. It is reported that uremic toxins may have direct neurotoxic (astrocyte activation and neuronal death) and/or indirect action through vascular effects (cerebral endothelial dysfunction, calcification, and inflammation). This review summarizes the evidence from research investigating the pathophysiological effects of phosphate toxicity in the nervous system, raising the question of whether the control of hyperphosphatemia in CKD would lower patients' risk of developing cognitive impairment and dementia.

Indexed as

Cerebrovascular DisordersCognitive DysfunctionDementiaRenal Insufficiency, ChronicHumansPhosphatesPhosphateschronic kidney diseasedementiaFGF23nervous systemphosphatePTH

Identifiers

PMID35806367
PMCPMC9266940
OpenAlexW4283801242

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.