Evidence mapPaperPMID 35938548Full record

ArticleRNA biology2022

Thrombosis-related circulating miR-16-5p is associated with disease severity in patients hospitalised for COVID-19.

Ceren Eyileten, Zofia Wicik, Sérgio N Simões, David C Martins-Jr, Krzysztof Klos, Wojciech Wlodarczyk, Alice Assinger, Dariusz Soldacki, Andrzej Chcialowski, Jolanta M Siller-Matula and 1 more

Open access · goldAbstract read
In one paragraph

Article in RNA biology, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed
2.1field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

20 citing papers in PubMed, 27 citations in OpenAlex.

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  13. RNA therapeutics.RNA biology · 2024
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 5 institutions in 3 countries.

Ceren EyiletenDepartment of Experimental and Clinical Pharmacology, Medical University of Warsaw, Center for Preclinical Research and Technology CEPT, Warsaw, Poland.ORCID 0000-0002-3324-9625
Zofia WicikDepartment of Experimental and Clinical Pharmacology, Medical University of Warsaw, Center for Preclinical Research and Technology CEPT, Warsaw, Poland.
Sérgio N SimõesDepartment of Informatics, Federal Institute of Espírito Santo, Serra, Brazil.
David C Martins-JrCenter for Mathematics, Computing and Cognition, Federal University of ABC, Santo André Brazil.
Krzysztof KlosDepartment of Infectious Diseases and Allergology - Military Institute of Medicine, Warsaw, Poland.
Wojciech WlodarczykDepartment of Infectious Diseases and Allergology - Military Institute of Medicine, Warsaw, Poland.
Alice AssingerDepartment of Vascular Biology and Thrombosis Research, Center of Physiology and Pharmacology, Medical University of Vienna, Austria.
Dariusz SoldackiDepartment of Clinical Immunology, Medical University of Warsaw, Warsaw, Poland.
Andrzej ChcialowskiDepartment of Infectious Diseases and Allergology - Military Institute of Medicine, Warsaw, Poland.
Jolanta M Siller-MatulaDepartment of Experimental and Clinical Pharmacology, Medical University of Warsaw, Center for Preclinical Research and Technology CEPT, Warsaw, Poland.
Marek PostulaDepartment of Experimental and Clinical Pharmacology, Medical University of Warsaw, Center for Preclinical Research and Technology CEPT, Warsaw, Poland.
Medical University of Warsaw · PLWojskowy Instytut Medycyny Lotniczej · PLInstituto Federal do Espírito Santo · BRMedical University of Vienna · ATUniversidade Federal do ABC · BR

Funding

Austrian Science Fund FWF P 34783
6 · The paper itself

Abstract

SARS-CoV-2 tropism for the ACE2 receptor, along with the multifaceted inflammatory reaction, is likely to drive the generalized hypercoagulable and thrombotic state seen in patients with COVID-19. Using the original bioinformatic workflow and network medicine approaches we reanalysed four coronavirus-related expression datasets and performed co-expression analysis focused on thrombosis and ACE2 related genes. We identified microRNAs (miRNAs) which play role in ACE2-related thrombosis in coronavirus infection and further, we validated the expressions of precisely selected miRNAs-related to thrombosis (miR-16-5p, miR-27a-3p, let-7b-5p and miR-155-5p) in 79 hospitalized COVID-19 patients and 32 healthy volunteers by qRT-PCR. Consequently, we aimed to unravel whether bioinformatic prioritization could guide selection of miRNAs with a potential of diagnostic and prognostic biomarkers associated with disease severity in patients hospitalized for COVID-19. In bioinformatic analysis, we identified EGFR, HSP90AA1, APP, TP53, PTEN, UBC, FN1, ELAVL1 and CALM1 as regulatory genes which could play a pivotal role in COVID-19 related thrombosis. We also found miR-16-5p, miR-27a-3p, let-7b-5p and miR-155-5p as regulators in the coagulation and thrombosis process.

Indexed as

COVID-19Induced Pluripotent Stem CellsMicroRNAsThrombosisAmyloid beta-PeptidesAngiotensin-Converting Enzyme 2BiomarkersHeat-Shock ProteinsHumansLeukocytes, MononuclearSARS-CoV-2Severity of Illness IndexAmyloid beta-PeptidesAngiotensin-Converting Enzyme 2BiomarkersHeat-Shock ProteinsMicroRNAsMIRN16 microRNA, humanACE2bioinformatics analysisin silico predictionmicroRNAsmiRNASARS-COV-2

Identifiers

PMID35938548
PMCPMC9361765
OpenAlexW4290613768

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.