Evidence map›Paper›PMID 35966894›Full record

ReviewThe World Allergy Organization journal2022

Environmental contributions to the interactions of COVID-19 and asthma: A secondary publication and update.

Marilyn Urrutia-Pereira, Herberto Jose Chong-Neto, Isabella Annesi Maesano, Ignacio J Ansotegui, Luis Caraballo, Lorenzo Cecchi, Carmen Galán, Juan Felipe López, Margarita Murrieta Aguttes, David Peden and 4 more

Open access · goldAbstract readReview
In one paragraph

Review in The World Allergy Organization journal, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
1.1field-weighted citation impact, top 26% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 10 citations in OpenAlex.

  1. Review
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

14 authors at 11 institutions in 6 countries.

Marilyn Urrutia-PereiraDepartment of Medicine, Federal University of Pampa, Uruguaiana, RS, Brazil.
Herberto Jose Chong-NetoDivision of Allergy and Immunology, Department of Pediatrics, Federal University of Paraná, Curitiba, PR, Brazil.
Isabella Annesi MaesanoFrench NIH (INSERM), and EPAR Department, IPLESP, INSERM and Sorbonne University, Paris, France.
Ignacio J AnsoteguiHospital Quironsalud Bizkaia, Bilbao-Erandio, Spain.
Luis CaraballoInstitute for Immunological Research, University of Cartagena, Cartagena, Colombia.
Lorenzo CecchiCentre of Bioclimatology, University of Florence, Florence, Italy.
Carmen GalánDepartment of Botany, Ecology and Plant Physiology, International Campus of Excellence on Agrifood (ceiA3), University of Córdoba, Córdoba, Spain.
Juan Felipe LópezInstitute for Immunological Research, University of Cartagena, Cartagena, Colombia.
Margarita Murrieta AguttesConsumer Healthcare Division, Sanofi-Aventis Group, Gentility, France.
David PedenUNC School of Medicine, University of North Carolina, Chapel Hill, NC, United States.
Anna PomésBasic Research, Indoor Biotechnologies, Inc, Charlottesville, VA, United States.
Josefina ZakzukInstitute for Immunological Research, University of Cartagena, Cartagena, Colombia.
Nelson A Rosário FilhoUniversity of Parana, Curitiba, PR, Brazil.
Gennaro D'AmatoDivision of Respiratory and Allergic Diseases, High Specialty Hospital A. Cardarelli, School of Specialization in Respiratory Diseases, Federico II University, Naples, Italy.
University of Cartagena · COAzienda Usl Toscana Centro · ITFederico II University Hospital · ITHospital Quirónsalud Barcelona · ESIndoor Biotechnologies (United States) · USInserm · FRSanofi (France) · FRUniversidade Federal do Pampa · BRUniversidade Federal do Paraná · BRUniversity of Córdoba · ESUniversity of North Carolina at Chapel Hill · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

An outbreak of coronavirus disease 2019 (COVID-19) caused by Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) started in Wuhan, Hubei Province, China and quickly spread around the world. Current evidence is contradictory on the association of asthma with COVID-19 and associated severe outcomes. Type 2 inflammation may reduce the risk for severe COVID-19. Whether asthma diagnosis may be a risk factor for severe COVID-19, especially for those with severe disease or non-allergic phenotypes, deserves further attention and clarification. In addition, COVID-19 does not appear to provoke asthma exacerbations, and asthma therapeutics should be continued for patients with exposure to COVID-19. Changes in the intensity of pollinization, an earlier start and extension of the pollinating season, and the increase in production and allergenicity of pollen are known direct effects that air pollution has on physical, chemical, and biological properties of the pollen grains. They are influenced and triggered by meteorological variables that could partially explain the effect on COVID-19. SARS-CoV-2 is capable of persisting in the environment and can be transported by bioaerosols which can further influence its transmission rate and seasonality. The COVID-19 pandemic has changed the behavior of adults and children globally. A general trend during the pandemic has been human isolation indoors due to school lockdowns and loss of job or implementation of virtual work at home. A consequence of this behavior change would presumably be changes in indoor allergen exposures and reduction of inhaled outdoor allergens. Therefore, lockdowns during the pandemic might have improved some specific allergies, while worsening others, depending on the housing conditions.

Indexed as

Allergic rhinitisAsthmaCOVID-19

Identifiers

PMID35966894
PMCPMC9359502
OpenAlexW4294354019

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.