Evidence mapPaperPMID 36090702Full record

ArticleInternational journal of general medicine2022

Independent Association of 25[OH]D Level on Reduced Glutathione and TNF-α in Patients ‎with Diabetes and/or Hypertension.

Faten A AlRadini, Azza A K El-Sheikh, Abeer S Al Shahrani, Norah M Alzamil, Amel A Fayed, Eman Alsayed, Shatha S Alharbi, Msaad A Altulihee, Shaimaa A Andijani, Wafa K AlShaiddi and 1 more

Open access · goldAbstract read
In one paragraph

Article in International journal of general medicine, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
0.1field-weighted citation impact, top 51% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 1 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 3 institutions in 3 countries.

Faten A AlRadiniDepartment of Clinical Sciences, College of Medicine, Princess Nourah bint ‎Abdulrahman University, Riyadh, Saudi Arabia.ORCID 0000-0002-7835-8681
Azza A K El-SheikhDepartment of Basic Sciences, College of Medicine, Princess Nourah bint Abdulrahman ‎University, Riyadh, Saudi Arabia.
Abeer S Al ShahraniDepartment of Clinical Sciences, College of Medicine, Princess Nourah bint ‎Abdulrahman University, Riyadh, Saudi Arabia.ORCID 0000-0003-1319-8936
Norah M AlzamilDepartment of Clinical Sciences, College of Medicine, Princess Nourah bint ‎Abdulrahman University, Riyadh, Saudi Arabia.ORCID 0000-0002-0609-738X
Amel A FayedDepartment of Clinical Sciences, College of Medicine, Princess Nourah bint ‎Abdulrahman University, Riyadh, Saudi Arabia.
Eman AlsayedDepartment of Clinical Pathology, Faculty of Medicine, Minia University, El-‎Minia, Egypt.ORCID 0000-0003-1004-6419
Shatha S AlharbiDepartment of Family and Community Health, King Abdullah bin Abdulaziz University ‎Hospital, Princess Nourah bint Abdulrahman University, Riyadh, Saudi Arabia.
Msaad A AltuliheeDepartment of Family and Community Health, King Abdullah bin Abdulaziz University ‎Hospital, Princess Nourah bint Abdulrahman University, Riyadh, Saudi Arabia.
Shaimaa A AndijaniDepartment of Family and Community Health, King Abdullah bin Abdulaziz University ‎Hospital, Princess Nourah bint Abdulrahman University, Riyadh, Saudi Arabia.
Wafa K AlShaiddiDepartment of Pathology and Laboratory Medicine, King Abdullah bin Abdulaziz ‎University Hospital, Princess Nourah bint Abdulrahman University, Riyadh, Saudi ‎Arabia.
Fahad A AlamriGlobal Center of Mass Gathering Medicine, Ministry of Health, Family Medicine, ‎Primary Health Center, Riyadh, Saudi Arabia.
Princess Nourah bint Abdulrahman University · SAMinia University · EGMinistry of Health · BN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Purpose: Oxidative and inflammatory pathways play a significant role in the pathophysiology of a wide variety of non-communicable diseases such as type 2 diabetes mellitus (T2DM) and hypertension. However, the effect of serum 25-hydroxyvitamin D ‎‎(25[OH]D) on these pathways is still controversial. To evaluate the association of 25[OH]D on ‎antioxidant and pro-inflammatory biomarkers, reduced glutathione (GSH) and tumor ‎necrosis factor (TNF)-α, in T2DM and hypertensive patients. Patients and Methods: This is a cross-sectional study of a consecutive sample of patients attending the the Family Medicine clinic at King Abdullah bin Abdulaziz University Hospital (KAAUH). Participants were screened for eligibility according to the following ‎criteria: aged above 18 years and diagnosed with T2DM and/or hypertension for at least one ‎year. Patients receiving any kind of vitamin D or calcium supplements within the last three ‎months were excluded, as were those with a history of ‎renal failure, cancer, liver, thyroid, or any other chronic inflammatory diseases. Results: In total 424 T2DM and/or ‎hypertensive patients (mean age 55±12 years) were recruited. In addition to routine ‎physical and laboratory examinations, levels of serum 25[OH]D, GSH and TNF-α were ‎measured. The prevalence of 25[OH]D deficiency (<50 nmol/L) was 35.1%, which was ‎independent from GSH and TNF-α levels. In T2DM, hypertensive and patients having ‎both diseases, GSH levels were 349.3±19, 355.4±19 and 428.8±20 μmol/L, ‎respectively. Uncontrolled T2DM and hypertension patients showed significantly higher ‎GSH compared with the controlled group. ‎Males showed slightly higher level ‎of TNF-α compared with females and uncontrolled hypertensive patients had relatively ‎higher TNF-α level when evaluated against controlled hypertensive patients. ‎. Conclusion: 25[OH]D level is independent of oxidative stress and inflammation, ‎assessed by levels of GSH and TNF-α, respectively, in T2DM and hypertensive Saudi ‎patients. ‎.

Indexed as

diabeteshypertensionreduced glutathioneTNF-αvitamin D

Identifiers

PMID36090702
PMCPMC9462432
OpenAlexW4294613069

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.