Evidence map›Paper›PMID 36096204›Full record

ArticleThe Journal of allergy and clinical immunology2023

Exacerbation-prone pediatric asthma is associated with arginine, lysine, and methionine pathway alterations.

Kirsten A Cottrill, Susan T Stephenson, Ahmad F Mohammad, Susan O Kim, Nael A McCarty, Rishikesan Kamaleswaran, Anne M Fitzpatrick, Joshua D Chandler

Open access · greenAbstract read
In one paragraph

Article in The Journal of allergy and clinical immunology, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
2.1field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

15 citing papers in PubMed, 19 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Metabolomic and immunological signatures of asthma severity in children.Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology · 2025
    Article
  5. Frequent exacerbator-a novel endotype of pediatric asthma.The Journal of allergy and clinical immunology · 2025
    Review
  6. Review
  7. Clinical and inflammatory features of traffic-related diesel exposure in children with asthma.Annals of allergy, asthma & immunology : official publication of the American College of Allergy, Asthma, & Immunology · 2024
    Article
  8. Article
  9. Article
  10. Amino acid metabolism in health and disease.Signal transduction and targeted therapy · 2023
    Review
  11. Article
  12. Article
  13. Article
  14. A prognosis prediction chromatin regulator signature for patients with severe asthma.Allergy, asthma, and clinical immunology : official journal of the Canadian Society of Allergy and Clinical Immunology · 2023
    Article
  15. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 2 institutions in 1 country.

Kirsten A CottrillDepartment of Pediatrics, Emory University, Atlanta, Ga.
Susan T StephensonDepartment of Pediatrics, Emory University, Atlanta, Ga.
Ahmad F MohammadDepartment of Pediatrics, Emory University, Atlanta, Ga.
Susan O KimDepartment of Pediatrics, Emory University, Atlanta, Ga.
Nael A McCartyDepartment of Pediatrics, Emory University, Atlanta, Ga.
Rishikesan KamaleswaranDepartment of Pediatrics, Emory University, Atlanta, Ga; Department of Biomedical Informatics, Emory University, Atlanta, Ga.
Anne M FitzpatrickDepartment of Pediatrics, Emory University, Atlanta, Ga; Children's Healthcare of Atlanta, Atlanta, Ga.
Joshua D ChandlerDepartment of Pediatrics, Emory University, Atlanta, Ga; Children's Healthcare of Atlanta, Atlanta, Ga. Electronic address: joshua.chandler@emory.edu.
Emory University · USChildren's Healthcare of Atlanta · US

Funding

Implementing a Maternal health and PRegnancy Outcomes Vision for Everyone (IMPROVE)UL1TR002378 · NCATS · EMORY UNIVERSITY · PI Andres J Garcia, Elizabeth O. Ofili · 2017 to 2026
$92.1M
Symptom clusters in children with exacerbation-prone asthmaR01NR018666 · NINR · EMORY UNIVERSITY · PI FITZPATRICK, ANNE MENTRO · 2019 to 2023
$3.1M
Training Program in Biochemistry, Cell and Molecular BiologyT32GM135060 · NIGMS · EMORY UNIVERSITY · PI Lawrence H. Boise, ANITA H. CORBETT · 2020 to 2026
$2.8M
Neutrophil hyperexocytosis and hypochlorous acid exposure in early cystic fibrosis lung diseaseR01HL150658 · NHLBI · EMORY UNIVERSITY · PI Joshua D Chandler · 2023 to 2026
$1.5M
Mentored patient-oriented research in preschool wheezing disordersK24NR018866 · NINR · EMORY UNIVERSITY · PI FITZPATRICK, ANNE MENTRO · 2020 to 2024
$625k
Neutrophil hyperexocytosis and hypochlorous acid exposure in early cystic fibrosis lung diseaseR56HL150658 · NHLBI · EMORY UNIVERSITY · PI CHANDLER, JOSHUA D · 2020 to 2020
$383k
NCATS NIH HHS UL1 TR002378NHLBI NIH HHS R01 HL150658NHLBI NIH HHS R56 HL150658NIGMS NIH HHS T32 GM135060NINR NIH HHS K24 NR018866NINR NIH HHS R01 NR018666
6 · The paper itself

Abstract

backgroundThe asthma of some children remains poorly controlled, with recurrent exacerbations despite treatment with inhaled corticosteroids. Aside from prior exacerbations, there are currently no reliable predictors of exacerbation-prone asthma in these children and only a limited understanding of the potential underlying mechanisms.

objectiveWe sought to quantify small molecules in the plasma of children with exacerbation-prone asthma through mass spectrometry-based metabolomics. We hypothesized that the plasma metabolome of these children would differ from that of children with non-exacerbation-prone asthma.

methodsPlasma metabolites were extracted from 4 pediatric asthma cohorts (215 total subjects, with 41 having exacerbation-prone asthma) and detected with a mass spectrometer. High-confidence annotations were retained for univariate analysis and were confirmed by a sensitivity analysis in subjects receiving high-dose inhaled corticosteroids. Metabolites that varied by cohort were excluded. MetaboAnalyst software was used to identify pathways of interest. Concentrations were calculated by reference standardization.

resultsWe identified 32 unique, cohort-independent metabolites that differed in children with exacerbation-prone asthma compared to children with non-exacerbation-prone asthma. Comparison of metabolite concentrations to literature-reported values for healthy children revealed that most metabolites were decreased in both asthma groups, but more so in exacerbation-prone asthma. Pathway analysis identified arginine, lysine, and methionine pathways as most impacted.

conclusionsSeveral plasma metabolites are perturbed in children with exacerbation-prone asthma and are largely related to arginine, lysine, and methionine pathways. While validation is needed, plasma metabolites may be potential biomarkers for exacerbation-prone asthma in children.

Indexed as

AsthmaLysineAdrenal Cortex HormonesArginineChildHumansMethionineRacemethionineAdrenal Cortex HormonesArginineLysineMethionineRacemethioninearginineAsthmaexacerbation pronelysinemetabolomicsmethioninepediatricplasma

Identifiers

PMID36096204
PMCPMC9825634
OpenAlexW4295021521

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.