Evidence map›Paper›PMID 36151065›Full record

ArticleJournal of neuroimaging : official journal of the American Society of Neuroimaging2022

Related amyloid burden and cortical atrophy in individuals with subtle cognitive decline.

Tess E K Cersonsky, Shanti Mechery, Louisa Thompson, Athene Lee, Jessica Alber, Indra Neil Sarkar, Leslie Ann D Brick

Open access · greenAbstract read
In one paragraph

Article in Journal of neuroimaging : official journal of the American Society of Neuroimaging, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.4field-weighted citation impact, top 38% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 3 citations in OpenAlex.

  1. Article
  2. Review
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 3 institutions in 1 country.

Tess E K CersonskyWarren Alpert Medical School of Brown University, Providence, Rhode Island, USA.
Shanti MecheryMemory and Aging Program, Butler Hospital, Providence, Rhode Island, USA.
Louisa ThompsonWarren Alpert Medical School of Brown University, Providence, Rhode Island, USA.
Athene LeeWarren Alpert Medical School of Brown University, Providence, Rhode Island, USA.
Jessica AlberMemory and Aging Program, Butler Hospital, Providence, Rhode Island, USA.
Indra Neil SarkarWarren Alpert Medical School of Brown University, Providence, Rhode Island, USA.
Leslie Ann D BrickWarren Alpert Medical School of Brown University, Providence, Rhode Island, USA.
Providence College · USBrown University · USUniversity of Rhode Island · US

Funding

Alzheimer's Disease Neuroimaging Initiative - SupplementU01AG024904 · NIA · NORTHERN CALIFORNIA INSTITUTE RES &EDUC · PI WEINER, MICHAEL W · 2004 to 2015
$121.0M
Tracking and Evaluation CoreU54GM115677 · NIGMS · BROWN UNIVERSITY · PI CHOUDHARY, GAURAV · 2016 to 2025
$45.0M
NIA NIH HHS U01 AG024904NIGMS NIH HHS U54 GM115677
6 · The paper itself

Abstract

background and purposeSubtle cognitive decline represents a stage of cognitive deterioration in which pathological biomarkers may be present, including early cortical atrophy and amyloid deposition. Using individual items from the Montreal Cognitive Assessment and k-modes cluster analysis, we previously identified three clusters of individuals without overt cognitive impairment: (1) High Performing (no deficits in performance), (2) Memory Deficits (lower memory performance), and (3) Compound Deficits (lower memory and executive function performance). In this study, we sought to understand the relationships found in our clusters between cortical atrophy on MR and amyloid burden on PET.

methodsData were derived from the Alzheimer's Disease Neuroimaging Initiative and comprised individuals from our previous analyses with available MR and amyloid PET scans (n = 272). Using multiple-group structural equation modeling, we regressed amyloid standardized uptake value ratio on volumetric regions to simultaneously evaluate unique associations within each cluster.

resultsIn our Compound Deficits cluster, greater whole cerebral amyloid burden was significantly related to right entorhinal cortical and left hippocampal atrophy, r

conclusionsThe Compound Deficits cluster, which represents a group potentially at higher risk for decline, was observed to have significantly more cortical atrophy, particularly within the entorhinal cortex and hippocampus, associated with whole brain and frontal lobe amyloid burden. These findings point to a pattern of early pathological deterioration that may place these individuals at risk for future decline.

Indexed as

Alzheimer DiseaseAmyloidosisCognitive DysfunctionAmyloidAmyloid beta-PeptidesAmyloidogenic ProteinsAtrophyBrainHumansMagnetic Resonance ImagingPositron-Emission TomographyAmyloidAmyloid beta-PeptidesAmyloidogenic ProteinsAlzheimer's diseaseamyloid burdencortical atrophyMontreal Cognitive Assessmentsubtle cognitive decline

Identifiers

PMID36151065
PMCPMC9903200
OpenAlexW4297037772

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.