Evidence map›Paper›PMID 36281949›Full record

ReviewOncology reports2022

Regulators of epigenetic change in ferroptosis‑associated cancer (Review).

Jiaming Wu, Shuang Zhu, Peng Wang, Jinge Wang, Jingjing Huang, Tong Wang, Lingfeng Guo, Desen Liang, Qinghui Meng, Huayang Pan

Open access · hybridAbstract readReview
In one paragraph

Review in Oncology reports, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
1.8field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 12 citations in OpenAlex.

  1. Review
  2. Article
  3. Review
  4. Review
  5. Review
  6. Review
  7. Review
  8. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 2 institutions in 1 country.

Jiaming Wu *Key Laboratory of Hepatosplenic Surgery, Department of General Surgery, Ministry of Education, First Affiliated Hospital of Harbin Medical University, Harbin Medical University, Harbin, Heilongjiang 150001, P.R. China.
Shuang Zhu *Department of Mental Health Institute, First Affiliated Hospital Medical University, Harbin Medical University, Harbin, Heilongjiang 150001, P.R. China.
Peng Wang *Key Laboratory of Hepatosplenic Surgery, Department of General Surgery, Ministry of Education, First Affiliated Hospital of Harbin Medical University, Harbin Medical University, Harbin, Heilongjiang 150001, P.R. China.
Jinge WangNursing Department, Second Affiliated Hospital of Harbin Medical University, Harbin Medical University, Harbin, Heilongjiang 150001, P.R. China.
Jingjing HuangKey Laboratory of Hepatosplenic Surgery, Department of General Surgery, Ministry of Education, First Affiliated Hospital of Harbin Medical University, Harbin Medical University, Harbin, Heilongjiang 150001, P.R. China.
Tong WangKey Laboratory of Hepatosplenic Surgery, Department of General Surgery, Ministry of Education, First Affiliated Hospital of Harbin Medical University, Harbin Medical University, Harbin, Heilongjiang 150001, P.R. China.
Lingfeng GuoKey Laboratory of Hepatosplenic Surgery, Department of General Surgery, Ministry of Education, First Affiliated Hospital of Harbin Medical University, Harbin Medical University, Harbin, Heilongjiang 150001, P.R. China.
Desen LiangKey Laboratory of Hepatosplenic Surgery, Department of General Surgery, Ministry of Education, First Affiliated Hospital of Harbin Medical University, Harbin Medical University, Harbin, Heilongjiang 150001, P.R. China.
Qinghui MengKey Laboratory of Hepatosplenic Surgery, Department of General Surgery, Ministry of Education, First Affiliated Hospital of Harbin Medical University, Harbin Medical University, Harbin, Heilongjiang 150001, P.R. China.
Huayang PanKey Laboratory of Hepatosplenic Surgery, Department of General Surgery, Ministry of Education, First Affiliated Hospital of Harbin Medical University, Harbin Medical University, Harbin, Heilongjiang 150001, P.R. China.
Harbin Medical University · CNFirst Affiliated Hospital of Harbin Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The occurrence of tumors is associated with the upregulation or downregulation of certain genes. The identification of novel tumor therapies has revealed that regulation of tumor cell death can either promote or suppress the occurrence and development of tumors. Iron‑dependent lipid free oxygen radical accumulation causes tumor cells to die by ferroptosis, a form of regulated cell death. Multiple mechanisms mediate this mode of cell death, including redox homeostasis, iron metabolism, mitochondrial activity, breakdown of amino acids, lipids and sugars and epigenetic regulatory and disease‑associated signaling pathways. The present review discussed epigenetic mechanism of ferroptosis with the aim of providing novel insight for optimization of the effects of antitumor therapy.

Indexed as

FerroptosisNeoplasmsAmino AcidsEpigenesis, GeneticHumansIronReactive Oxygen SpeciesSugarsAmino AcidsIronReactive Oxygen SpeciesSugarscancerepigeneticferroptosis

Identifiers

PMID36281949
PMCPMC9641706
OpenAlexW4306878700

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.