Evidence mapPaperPMID 36306151Full record

Trial reportDiabetes, obesity & metabolism2023

Comparative effects of weight loss and incretin-based therapies on vascular endothelial function, fibrinolysis and inflammation in individuals with obesity and prediabetes: A randomized controlled trial.

Mona Mashayekhi, Joshua A Beckman, Hui Nian, Erica M Garner, Dustin Mayfield, Jessica K Devin, John R Koethe, Jonathan D Brown, Katherine N Cahill, Chang Yu and 4 more

Open access · greenAbstract readRandomized Controlled Trial
In one paragraph

Trial report in Diabetes, obesity & metabolism, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 26 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
26citing papers in PubMed, 2 pooled it
4.7field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

26 citing papers in PubMed, 2 syntheses or guidelines pooled it, 34 citations in OpenAlex.

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  15. Inflammation and resolution in obesity.Nature reviews. Endocrinology · 2025 · on this map
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

14 authors at 6 institutions in 1 country.

Mona MashayekhiDepartment of Medicine, Division of Diabetes, Endocrinology and Metabolism, Vanderbilt University Medical Center, Nashville, Tennessee.ORCID 0000-0003-0331-2395
Joshua A BeckmanDepartment of Medicine, Division of Vascular Medicine, UTSouthwestern, Dallas, Texas.ORCID 0000-0001-8332-8439
Hui NianDepartment of Biostatistics, Vanderbilt University Medical Center, Nashville, Tennessee.
Erica M GarnerDepartment of Medicine, Division of Diabetes, Endocrinology and Metabolism, Vanderbilt University Medical Center, Nashville, Tennessee.
Dustin MayfieldDepartment of Medicine, Division of Clinical Pharmacology, Vanderbilt University Medical Center, Nashville, Tennessee.
Jessica K DevinUCHealth Endocrinology, Yampa Valley Medical Center, Steamboat Springs, Colorado.
John R KoetheVeterans Affairs Tennessee Valley Healthcare System, Nashville, Tennessee.
Jonathan D BrownDepartment of Medicine, Division of Cardiovascular Medicine, Vanderbilt University Medical Center, Nashville, Tennessee.
Katherine N CahillDepartment of Medicine, Division of Allergy, Pulmonary and Critical Care Medicine, Vanderbilt University Medical Center, Nashville, Tennessee.
Chang YuDepartment of Population Health, NYU Grossman School of Medicine, New York, New York.
Heidi SilverVeterans Affairs Tennessee Valley Healthcare System, Nashville, Tennessee.
Kevin NiswenderDepartment of Medicine, Division of Diabetes, Endocrinology and Metabolism, Vanderbilt University Medical Center, Nashville, Tennessee.
James M LutherDepartment of Medicine, Division of Clinical Pharmacology, Vanderbilt University Medical Center, Nashville, Tennessee.
Nancy J BrownYale School of Medicine, New Haven, Connecticut.
Vanderbilt University Medical Center · USVA Tennessee Valley Healthcare System · USNew York University · USSouthwestern Medical Center · USUCHealth Yampa Valley Medical Center · USYale University · US

Funding

Vanderbilt Institute for Clinical and Translational Research (VICTR)UL1TR002243 · VANDERBILT UNIVERSITY MEDICAL CENTER · 2025 to 2025
$10.7M
Glucagon-Like Peptide-1 Receptor Agonist Treatment in Adult, Obesity-Related, Symptomatic AsthmaU01AI155299 · VANDERBILT UNIVERSITY MEDICAL CENTER · 2025 to 2025
$2.3M
RESEARCH TRAINING IN DIABETICS AND ENDOCRINOLOGYT32DK007061 · VANDERBILT UNIVERSITY MEDICAL CENTER · 1986 to 2025
$1.8M
Vanderbilt Diabetes Research CenterP30DK020593 · VANDERBILT UNIVERSITY MEDICAL CENTER · 2025 to 2025
$1.8M
Viral and Host Determinants of Infant and Childhood Allergy and AsthmaU19AI095227 · VANDERBILT UNIVERSITY MEDICAL CENTER · 2025 to 2025
$1.5M
NCATS NIH HHS UL1 TR000445NCATS NIH HHS UL1 TR002243NHLBI NIH HHS R01 HL125426NHLBI NIH HHS R01 HL146654NIAID NIH HHS U01 AI155299NIAID NIH HHS U19 AI095227NIDDK NIH HHS P30 DK020593NIDDK NIH HHS T32 DK007061
6 · The paper itself

Abstract

aimTo test the hypothesis that glucagon-like peptide-1 receptor (GLP-1R) agonists have beneficial effects on vascular endothelial function, fibrinolysis and inflammation through weight loss-independent mechanisms. MATERIALS AND

methodsIndividuals with obesity and prediabetes were randomized to 14 weeks of the GLP-1R agonist liraglutide, hypocaloric diet or the dipeptidyl peptidase-4 inhibitor sitagliptin in a 2:1:1 ratio. Treatment with drug was double blind and placebo-controlled. Measurements were made at baseline, after 2 weeks prior to significant weight loss and after 14 weeks. The primary outcomes were measures of endothelial function: flow-mediated vasodilation (FMD), plasminogen activator inhibitor-1 (PAI-1) and urine albumin-to-creatinine ratio (UACR).

resultsEighty-eight individuals were studied (liraglutide N = 44, diet N = 22, sitagliptin N = 22). Liraglutide and diet reduced weight, insulin resistance and PAI-1, while sitagliptin did not. There was no significant effect of any treatment on endothelial vasodilator function measured by FMD. Post hoc subgroup analyses in individuals with baseline FMD below the median, indicative of greater endothelial dysfunction, showed an improvement in FMD by all three treatments. GLP-1R antagonism with exendin (9-39) increased fasting blood glucose but did not change FMD or PAI-1. There was no effect of treatment on UACR. Finally, liraglutide, but not sitagliptin or diet, reduced the chemokine monocyte chemoattractant protein-1 (MCP-1).

conclusionLiraglutide and diet reduce weight, insulin resistance and PAI-1. Liraglutide, sitagliptin and diet do not change FMD in obese individuals with prediabetes with normal endothelial function. Liraglutide alone lowers the pro-inflammatory and pro-atherosclerotic chemokine MCP-1, indicating that this beneficial effect is independent of weight loss.

Indexed as

Insulin ResistancePrediabetic StateDiet, ReducingFibrinolysisGlucagon-Like Peptide-1 Receptor AgonistsHumansHypoglycemic AgentsIncretinsInflammationLiraglutideObesityPlasminogen Activator Inhibitor 1Sitagliptin PhosphateWeight LossGlucagon-Like Peptide-1 Receptor AgonistsHypoglycemic AgentsIncretinsLiraglutidePlasminogen Activator Inhibitor 1Sitagliptin Phosphatecardiovascular diseasedietary interventionDPP4 inhibitorGLP-1 receptor agonistincretin physiologyrandomized trial

Identifiers

PMID36306151
PMCPMC10306232
OpenAlexW4307498814

What Socratic holds

Texttitle and abstract
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.