Evidence map›Paper›PMID 36358919›Full record

ReviewBiomolecules2022

Focusing on the Role of Natural Products in Overcoming Cancer Drug Resistance: An Autophagy-Based Perspective.

Jiaqi Yao, Chi Ma, Kaixuan Feng, Guang Tan, Qingping Wen

Open access · goldAbstract readReview
In one paragraph

Review in Biomolecules, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
2.5field-weighted citation impact, top 10% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 17 citations in OpenAlex.

  1. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

Jiaqi YaoDepartment of Anesthesiology, The First Affiliated Hospital of Dalian Medical University, Dalian 116011, China.
Chi MaDepartment of Hepatobiliary Surgery, The First Affiliated Hospital of Dalian Medical University, Dalian 116011, China.
Kaixuan FengDepartment of Anesthesiology, The Affiliated Xinhua Hospital of Dalian University, Dalian 116021, China.
Guang TanDepartment of Hepatobiliary Surgery, The First Affiliated Hospital of Dalian Medical University, Dalian 116011, China.
Qingping WenDepartment of Anesthesiology, The First Affiliated Hospital of Dalian Medical University, Dalian 116011, China.ORCID 0000-0002-0856-9846
Dalian Medical University · CNDalian University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Autophagy is a critical cellular adaptive response in tumor formation. Nutritional deficiency and hypoxia exacerbate autophagic flux in established malignancies, promoting tumor cell proliferation, migration, metastasis, and resistance to therapeutic interventions. Pro-survival autophagy inhibition may be a promising treatment option for advanced cancer. Furthermore, excessive or persistent autophagy is cytotoxic, resulting in tumor cell death. Targeted autophagy activation has also shown significant promise in the fight against tumor drug resistance. Several research groups have examined the ability of natural products (NPs) such as alkaloids, terpenoids, polyphenols, and anthraquinones to serve as autophagy inhibitors or activators. The data support the capacity of NPs that promote lethal autophagy or inhibit pro-survival autophagy from being employed against tumor drug resistance. This paper discusses the potential applications of NPs that regulate autophagy in the fight against tumor drug resistance, some limitations of the current studies, and future research needs and priorities.

Indexed as

Antineoplastic AgentsBiological ProductsNeoplasmsAutophagyCell Line, TumorDrug Resistance, NeoplasmHumansAntineoplastic AgentsBiological Productsautophagynatural productsresistancetherapytumors

Identifiers

PMID36358919
PMCPMC9687214
OpenAlexW4307553143

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.