Evidence map›Paper›PMID 36432618›Full record

ReviewNutrients2022

Molecular Mechanisms for Ketone Body Metabolism, Signaling Functions, and Therapeutic Potential in Cancer.

Chi Yeon Hwang, Wonchae Choe, Kyung-Sik Yoon, Joohun Ha, Sung Soo Kim, Eui-Ju Yeo, Insug Kang

Open access · goldAbstract readReview
In one paragraph

Review in Nutrients, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 63 papers.

0numbers the graph read from it
0cells of the map it votes in
63citing papers in PubMed
10.1field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

63 citing papers in PubMed, 95 citations in OpenAlex.

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3 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 1 country.

Chi Yeon HwangDepartment of Biomedical Sciences, Graduate School, Kyung Hee University, Seoul 02447, Republic of Korea.
Wonchae ChoeDepartment of Biomedical Sciences, Graduate School, Kyung Hee University, Seoul 02447, Republic of Korea.ORCID 0000-0001-5764-9482
Kyung-Sik YoonDepartment of Biomedical Sciences, Graduate School, Kyung Hee University, Seoul 02447, Republic of Korea.ORCID 0000-0002-2473-4493
Joohun HaDepartment of Biomedical Sciences, Graduate School, Kyung Hee University, Seoul 02447, Republic of Korea.
Sung Soo KimDepartment of Biomedical Sciences, Graduate School, Kyung Hee University, Seoul 02447, Republic of Korea.
Eui-Ju YeoDepartment of Biochemistry, College of Medicine, Gachon University, Incheon 21999, Republic of Korea.
Insug KangDepartment of Biomedical Sciences, Graduate School, Kyung Hee University, Seoul 02447, Republic of Korea.ORCID 0000-0001-7117-1008
Kyung Hee University · KRGachon University · KR

Funding

the National Research Foundation of Korea 2021R1F1A1049054 and 2018R1A6A1A03025124
6 · The paper itself

Abstract

The ketone bodies (KBs) β-hydroxybutyrate and acetoacetate are important alternative energy sources for glucose during nutrient deprivation. KBs synthesized by hepatic ketogenesis are catabolized to acetyl-CoA through ketolysis in extrahepatic tissues, followed by the tricarboxylic acid cycle and electron transport chain for ATP production. Ketogenesis and ketolysis are regulated by the key rate-limiting enzymes, 3-hydroxy-3-methylglutaryl-CoA synthase 2 and succinyl-CoA:3-oxoacid-CoA transferase, respectively. KBs participate in various cellular processes as signaling molecules. KBs bind to G protein-coupled receptors. The most abundant KB, β-hydroxybutyrate, regulates gene expression and other cellular functions by inducing post-translational modifications. KBs protect tissues by regulating inflammation and oxidative stress. Recently, interest in KBs has been increasing due to their potential for treatment of various diseases such as neurological and cardiovascular diseases and cancer. Cancer cells reprogram their metabolism to maintain rapid cell growth and proliferation. Dysregulation of KB metabolism also plays a role in tumorigenesis in various types of cancer. Targeting metabolic changes through dietary interventions, including fasting and ketogenic diets, has shown beneficial effects in cancer therapy. Here, we review current knowledge of the molecular mechanisms involved in the regulation of KB metabolism and cellular signaling functions, and the therapeutic potential of KBs and ketogenic diets in cancer.

Indexed as

Diet, KetogenicNeoplasms3-Hydroxybutyric AcidHumansKetone BodiesSignal Transduction3-Hydroxybutyric AcidKetone Bodiescancerinflammationketogenic dietketone bodiesoxidative stresspost-translational modificationsβ-hydroxybutyrate

Identifiers

PMID36432618
PMCPMC9694619
OpenAlexW4309717806

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.