Evidence map›Paper›PMID 36598607›Full record

ArticleHandbook of experimental pharmacology2023

Mechanisms of Vascular Ca

Miguel Martín-Aragón Baudel, Junyoung Hong, Johannes W Hell, Madeline Nieves-Cintrón, Manuel F Navedo

Abstract read
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In one paragraph

Article in Handbook of experimental pharmacology, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
4.2field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 5 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Miguel Martín-Aragón BaudelDepartment of Pharmacology, University of California Davis, Davis, CA, USA. martinaragon@ucdavis.edu.
Junyoung HongDepartment of Pharmacology, University of California Davis, Davis, CA, USA.
Johannes W HellDepartment of Pharmacology, University of California Davis, Davis, CA, USA.
Madeline Nieves-CintrónDepartment of Pharmacology, University of California Davis, Davis, CA, USA.
Manuel F NavedoDepartment of Pharmacology, University of California Davis, Davis, CA, USA. mfnavedo@ucdavis.edu.
University of California, Davis · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Diabetes is a leading cause of disability and mortality worldwide. A major underlying factor in diabetes is the excessive glucose levels in the bloodstream (e.g., hyperglycemia). Vascular complications directly result from this metabolic abnormality, leading to disabling and life-threatening conditions. Dysfunction of vascular smooth muscle cells is a well-recognized factor mediating vascular complications during diabetic hyperglycemia. The function of vascular smooth muscle cells is exquisitely controlled by different ion channels. Among the ion channels, the L-type Ca

Indexed as

Diabetes MellitusHyperglycemiaA Kinase Anchor ProteinsCalcium Channels, L-TypeHumansMuscle, Smooth, VascularMyocytes, Smooth MuscleAKAP5 protein, humanA Kinase Anchor ProteinsCalcium Channels, L-TypeL-type calcium channel alpha(1C)CalciumDiabetesIon channelSmooth muscleVasculature

Identifiers

PMID36598607
OpenAlexW4313488540

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.