Evidence mapPaperPMID 36734166Full record

Trial reportThe Journal of clinical endocrinology and metabolism2023

Pre-Meal Whey Protein Alters Postprandial Insulinemia by Enhancing β-Cell Function and Reducing Insulin Clearance in T2D.

Kieran Smith, Guy S Taylor, Mark Walker, Lise H Brunsgaard, Kelly A Bowden Davies, Emma J Stevenson, Daniel J West

Abstract readRandomized Controlled Trial
In one paragraph

Trial report in The Journal of clinical endocrinology and metabolism, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed, 1 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Trial
  3. Trial
  4. Article
  5. Therapeutic Potential of White Kidney Beans (Foods (Basel, Switzerland) · 2025
    Review
  6. Review
  7. Review
  8. Article
  9. Review
  10. Manipulation of Post-Prandial Hyperglycaemia in Type 2 Diabetes: An Update for Practitioners.Diabetes, metabolic syndrome and obesity : targets and therapy · 2024
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Kieran SmithPopulation Health Sciences Institute, Newcastle University, Newcastle upon Tyne NE1 7RU, UK.ORCID 0000-0001-7275-4379
Guy S TaylorPopulation Health Sciences Institute, Newcastle University, Newcastle upon Tyne NE1 7RU, UK.
Mark WalkerBiosciences Institute, Newcastle University, Newcastle upon Tyne NE2 4HH, UK.ORCID 0000-0002-2526-9810
Lise H BrunsgaardHealth and Performance Nutrition, Arla Foods Ingredients Group P/S, Viby J 8260, Denmark.
Kelly A Bowden DaviesPopulation Health Sciences Institute, Newcastle University, Newcastle upon Tyne NE1 7RU, UK.
Emma J StevensonPopulation Health Sciences Institute, Newcastle University, Newcastle upon Tyne NE1 7RU, UK.
Daniel J WestPopulation Health Sciences Institute, Newcastle University, Newcastle upon Tyne NE1 7RU, UK.ORCID 0000-0003-2246-4925

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

contextTreatments that reduce postprandial glycemia (PPG) independent of stimulating insulin secretion are appealing for the management of type 2 diabetes (T2D). Consuming pre-meal whey protein (WP) reduces PPG by delaying gastric emptying and increasing plasma insulin concentrations. However, its effects on β-cell function and insulin kinetics remains unclear.

objectiveTo examine the PPG-regulatory effects of pre-meal WP by modeling insulin secretion rates (ISR), insulin clearance, and β-cell function.

methodsThis was a single-blind, randomized, placebo-controlled, crossover design study in 18 adults with T2D (HbA1c, 56.7 ± 8.8 mmol/mol) who underwent 2 240-minute mixed-meal tolerance tests. Participants consumed WP (15 g protein) or placebo (0 g protein) 10 minutes before a mixed-macronutrient breakfast meal. PPG, pancreatic islet, and incretin hormones were measured throughout. ISR was calculated by C-peptide deconvolution. Estimates of insulin clearance and β-cell function were modeled from glucose, insulin, and ISR. Changes in PPG incremental area under the curve (iAUC; prespecified) and insulin clearance (post hoc) were measured.

resultsβ-cell function was 40% greater after WP (P = .001) and was accompanied with a -22% reduction in postprandial insulin clearance vs placebo (P < .0001). Both the peak change and PPG iAUC were reduced by WP (-1.5 mmol/L and -16%, respectively; both P < .05). Pre-meal WP augmented a 5.9-fold increase in glucagon and glucagon-like peptide 1 iAUC (both P < .0001), and a 1.5-fold increase in insulin iAUC (P < .001). Although the plasma insulin response was greater following WP, ISR was unaffected (P = .133).

conclusionIn adults with T2D, pre-meal WP reduced PPG by coordinating an enhancement in β-cell function with a reduction in insulin clearance. This enabled an efficient postprandial insulinemic profile to be achieved without requiring further β-cell stimulation.Trial registry ISRCTN ID: ISRCTN17563146 Website link: www.isrctn.com/ISRCTN17563146.

Indexed as

Diabetes Mellitus, Type 2InsulinAdultBlood GlucoseCross-Over StudiesHumansKineticsPostprandial PeriodSingle-Blind MethodWhey ProteinsBlood GlucoseInsulinWhey ProteinsGLP-1incretininsulin clearancePostprandial hyperglycemiatype 2 diabetes

Identifiers

PMID36734166
PMCPMC10807909

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.