Evidence map›Paper›PMID 36829792›Full record

ArticleAntioxidants (Basel, Switzerland)2023

IGF-1 Controls Metabolic Homeostasis and Survival in HEI-OC1 Auditory Cells through AKT and mTOR Signaling.

Ángela García-Mato, Blanca Cervantes, Lourdes Rodríguez-de la Rosa, Isabel Varela-Nieto

Open access · goldAbstract read
In one paragraph

Article in Antioxidants (Basel, Switzerland), 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
3.5field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed, 35 citations in OpenAlex.

  1. Review
  2. Review
  3. Autophagy in Sensorineural Hearing Loss: Jekyll or Hyde?International journal of molecular sciences · 2026
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 2 countries.

Ángela García-MatoNeuropathology of Hearing and Myelinopathies Group, Institute for Biomedical Research "Alberto Sols", Spanish National Research Council-Autonomous University of Madrid (CSIC-UAM), 28029 Madrid, Spain.ORCID 0000-0001-9961-8377
Blanca CervantesNeuropathology of Hearing and Myelinopathies Group, Institute for Biomedical Research "Alberto Sols", Spanish National Research Council-Autonomous University of Madrid (CSIC-UAM), 28029 Madrid, Spain.
Lourdes Rodríguez-de la RosaConsorcio Centro de Investigación Biomédica en Red (CIBERER), Institute of Health Carlos III (ISCIII), 28029 Madrid, Spain.ORCID 0000-0002-7518-6036
Isabel Varela-NietoNeuropathology of Hearing and Myelinopathies Group, Institute for Biomedical Research "Alberto Sols", Spanish National Research Council-Autonomous University of Madrid (CSIC-UAM), 28029 Madrid, Spain.ORCID 0000-0003-3077-0500
Consejo Superior de Investigaciones Científicas · ESInstituto de Salud Carlos III · ES

Funding

POCTEP FGCSIC/ PSL-INTERREG/FEDER 0551_PSL_6_E NITROPROHEARSpanish MCIN 501100011033 THEARPY-PID2020-115274RB-I00
6 · The paper itself

Abstract

Insulin-like growth factor 1 (IGF-1) is a trophic factor for the nervous system where it exerts pleiotropic effects, including the regulation of metabolic homeostasis. IGF-1 deficiency induces morphological alterations in the cochlea, apoptosis and hearing loss. While multiple studies have addressed the role of IGF-1 in hearing protection, its potential function in the modulation of otic metabolism remains unclear. Here, we report that "House Ear Institute-organ of Corti 1" (HEI-OC1) auditory cells express IGF-system genes that are regulated during their differentiation. Upon binding to its high-affinity receptor IGF1R, IGF-1 activates AKT and mTOR signaling to stimulate anabolism and, concomitantly, to reduce autophagic catabolism in HEI-OC1 progenitor cells. Notably, IGF-1 stimulation during HEI-OC1 differentiation to mature otic cells sustained both constructive metabolism and autophagic flux, possibly to favor cell remodeling. IGF1R engagement and downstream AKT signaling promoted HEI-OC1 cell survival by maintaining redox balance, even when cells were challenged with the ototoxic agent cisplatin. Our findings establish that IGF-1 not only serves an important function in otic metabolic homeostasis but also activates antioxidant defense mechanisms to promote hair cell survival during the stress response to insults.

Indexed as

anabolismapoptosisautophagycisplatinIGF1RNRF2otic differentiationoxidative stress

Identifiers

PMID36829792
PMCPMC9952701
OpenAlexW4317502686

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.