Evidence mapPaperPMID 36830880Full record

ArticleBiomedicines2023

SDF-1α-Releasing Microspheres Effectively Extend Stem Cell Homing after Myocardial Infarction.

Karolina Bajdak-Rusinek, Agnieszka Fus-Kujawa, Piotr Buszman, Dorota Żyła-Uklejewicz, Katarzyna Jelonek, Monika Musiał-Kulik, Carlos Fernandez, Magdalena Michalak, Kurian George, Janusz Kasperczyk and 1 more

Open access · goldAbstract read
In one paragraph

Article in Biomedicines, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
1.1field-weighted citation impact, top 22% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 5 citations in OpenAlex.

  1. Article
  2. Review
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 3 institutions in 1 country.

Karolina Bajdak-RusinekDepartment of Medical Genetics, Faculty of Medical Sciences in Katowice, Medical University of Silesia, 40-752 Katowice, Poland.ORCID 0000-0002-8235-5627
Agnieszka Fus-KujawaDepartment of Medical Genetics, Faculty of Medical Sciences in Katowice, Medical University of Silesia, 40-752 Katowice, Poland.ORCID 0000-0003-1119-4253
Piotr BuszmanCardiology Department, Andrzej Frycz Modrzewski Krakow University, 30-705 Krakow, Poland.
Dorota Żyła-UklejewiczCenter for Cardiovascular Research and Development, American Heart of Poland, 40-028 Katowice, Poland.
Katarzyna JelonekCentre of Polymer and Carbon Materials, Polish Academy of Sciences, 41-819 Zabrze, Poland.ORCID 0000-0003-2625-5434
Monika Musiał-KulikCentre of Polymer and Carbon Materials, Polish Academy of Sciences, 41-819 Zabrze, Poland.ORCID 0000-0001-6094-4677
Carlos FernandezCenter for Cardiovascular Research and Development, American Heart of Poland, 40-028 Katowice, Poland.
Magdalena MichalakCenter for Cardiovascular Research and Development, American Heart of Poland, 40-028 Katowice, Poland.
Kurian GeorgeDepartment of Medical Genetics, Faculty of Medical Sciences in Katowice, Medical University of Silesia, 40-752 Katowice, Poland.
Janusz KasperczykCentre of Polymer and Carbon Materials, Polish Academy of Sciences, 41-819 Zabrze, Poland.ORCID 0000-0002-8943-9508
Paweł BuszmanCenter for Cardiovascular Research and Development, American Heart of Poland, 40-028 Katowice, Poland.
Medical University of Silesia · PLAmerican Heart of Poland · PLCentre of Polymer and Carbon Materials · PL

Funding

National Science Center UMO- UMO - 2016/23/B/NZ5/02517
6 · The paper itself

Abstract

Ischemic heart disease (IHD) is one of the main focuses in today's healthcare due to its implications and complications, and it is predicted to be increasing in prevalence due to the ageing population. Although the conventional pharmacological and interventional methods for the treatment of IHD presents with success in the clinical setting, the long-term complications of cardiac insufficiency are on a continual incline as a result of post-infarction remodeling of the cardiac tissue. The migration and involvement of stem cells to the cardiac muscle, followed by differentiation into cardiac myocytes, has been proven to be the natural process, though at a slow rate. SDF-1α is a novel candidate to mobilize stem cells homing to the ischemic heart. Endogenous SDF-1α levels are elevated after myocardial infarction, but their presence gradually decreases after approximately seven days. Additional administration of SDF-1α-releasing microspheres could be a tool for the extension of the time the stem cells are in the cardiac tissue after myocardial infarction. This, in turn, could constitute a novel therapy for more efficient regeneration of the heart muscle after injury. Through this practical study, it has been shown that the controlled release of SDF-1α from biodegradable microspheres into the pericardial sac fourteen days after myocardial infarction increases the concentration of exogenous SDF-1α, which persists in the tissue much longer than the level of endogenous SDF-1α. In addition, administration of SDF-1α-releasing microspheres increased the expression of the factors potentially involved in the involvement and retention of myocardial stem cells, which constitutes vascular endothelial growth factor A (VEGFA), stem cell factor (SCF), and vascular cell adhesion molecules (VCAMs) at the site of damaged tissue. This exhibits the possibility of combating the basic limitations of cell therapy, including ineffective stem cell implantation and the ability to induce the migration of endogenous stem cells to the ischemic cardiac tissue and promote heart repair.

Indexed as

biodegradable polymeric microspheresbone marrow mesenchymal stromal cells (bmMSCs)myocardial infarctionSDF-1α

Identifiers

PMID36830880
PMCPMC9953248
OpenAlexW4318052507

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.