Evidence map›Paper›PMID 36834757›Full record

ArticleInternational journal of molecular sciences2023

CB2 Receptor as Emerging Anti-Inflammatory Target in Duchenne Muscular Dystrophy.

Maura Argenziano, Vincenzo Pota, Alessandra Di Paola, Chiara Tortora, Maria Maddalena Marrapodi, Giulia Giliberti, Domenico Roberti, Maria Caterina Pace, Francesca Rossi

Open access · goldAbstract read
In one paragraph

Article in International journal of molecular sciences, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
1.5field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 10 citations in OpenAlex.

  1. Review
  2. Review
  3. Article
  4. Article
  5. Article
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  9. Article
  10. Molecular Advances on Cannabinoid and Endocannabinoid Research.International journal of molecular sciences · 2023
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 1 institution in 1 country.

Maura ArgenzianoDepartment of Woman, Child and General and Specialistic Surgery, University of Campania "Luigi Vanvitelli", Via Luigi De Crecchio, 80138 Naples, Italy.
Vincenzo PotaDepartment of Woman, Child and General and Specialistic Surgery, University of Campania "Luigi Vanvitelli", Via Luigi De Crecchio, 80138 Naples, Italy.ORCID 0000-0001-9999-3388
Alessandra Di PaolaDepartment of Woman, Child and General and Specialistic Surgery, University of Campania "Luigi Vanvitelli", Via Luigi De Crecchio, 80138 Naples, Italy.
Chiara TortoraDepartment of Woman, Child and General and Specialistic Surgery, University of Campania "Luigi Vanvitelli", Via Luigi De Crecchio, 80138 Naples, Italy.
Maria Maddalena MarrapodiDepartment of Woman, Child and General and Specialistic Surgery, University of Campania "Luigi Vanvitelli", Via Luigi De Crecchio, 80138 Naples, Italy.
Giulia GilibertiDepartment of Experimental Medicine, University of Campania "Luigi Vanvitelli", Via Luigi De Crecchio, 80138 Naples, Italy.
Domenico RobertiDepartment of Woman, Child and General and Specialistic Surgery, University of Campania "Luigi Vanvitelli", Via Luigi De Crecchio, 80138 Naples, Italy.ORCID 0000-0003-0948-3910
Maria Caterina PaceDepartment of Woman, Child and General and Specialistic Surgery, University of Campania "Luigi Vanvitelli", Via Luigi De Crecchio, 80138 Naples, Italy.
Francesca RossiDepartment of Woman, Child and General and Specialistic Surgery, University of Campania "Luigi Vanvitelli", Via Luigi De Crecchio, 80138 Naples, Italy.
University of Campania "Luigi Vanvitelli" · IT

Funding

Ministero dell'Università e della Ricerca PRIN (Progetti di Rilevante Interesse Nazionale) 2017
6 · The paper itself

Abstract

Duchenne Muscular Dystrophy (DMD) is a very severe X-linked dystrophinopathy. It is due to a mutation in the DMD gene and causes muscular degeneration in conjunction with several secondary co-morbidities, such cardiomyopathy and respiratory failure. DMD is characterized by a chronic inflammatory state, and corticosteroids represent the main therapy for these patients. To contradict drug-related side effects, there is need for novel and more safe therapeutic strategies. Macrophages are immune cells stringently involved in both physiological and pathological inflammatory processes. They express the CB2 receptor, one of the main elements of the endocannabinoid system, and have been proposed as an anti-inflammatory target in several inflammatory and immune diseases. We observed a lower expression of the CB2 receptor in DMD-associated macrophages, hypothesizing its involvement in the pathogenesis of this pathology. Therefore, we analyzed the effect of JWH-133, a CB2 receptor selective agonist, on DMD-associated primary macrophages. Our study describes the beneficial effect of JWH-133 in counteracting inflammation by inhibiting pro-inflammatory cytokines release and by directing macrophages' phenotype toward the M2 anti-inflammatory one.

Indexed as

CardiomyopathiesMuscular Dystrophy, DuchenneAnti-Inflammatory AgentsCannabinoidsHumansInflammationReceptor, Cannabinoid, CB21,1-dimethylbutyl-1-deoxy-Delta(9)-THCAnti-Inflammatory AgentsCannabinoidsCNR2 protein, humanReceptor, Cannabinoid, CB2CB2 receptorDuchenne muscular dystrophyinflammationmacrophage phenotype

Identifiers

PMID36834757
PMCPMC9964283
OpenAlexW4319600714

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.