Evidence mapPaperPMID 36902001Full record

ArticleInternational journal of molecular sciences2023

Compositional Alteration of Gut Microbiota in Psoriasis Treated with IL-23 and IL-17 Inhibitors.

Yu-Huei Huang, Lun-Ching Chang, Ya-Ching Chang, Wen-Hung Chung, Shun-Fa Yang, Shih-Chi Su

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Article in International journal of molecular sciences, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
18citing papers in PubMed, 2 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

18 citing papers in PubMed, 2 syntheses or guidelines pooled it.

  1. Pooled it
  2. Pooled it
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  10. Observational
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  13. Gut microbiota and psoriasis: pathogenesis, targeted therapy, and future directions.Frontiers in cellular and infection microbiology · 2024
    Review
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  15. Article
  16. Article
  17. Hepatoprotective Potential ofIn vivo (Athens, Greece)
    Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Yu-Huei HuangInstitute of Medicine, Chung Shan Medical University, Taichung 402, Taiwan.ORCID 0000-0003-0574-1839
Lun-Ching ChangDepartment of Mathematical Sciences, Florida Atlantic University, Boca Raton, FL 33431, USA.ORCID 0000-0002-8039-5350
Ya-Ching ChangDepartment of Dermatology, Chang Gung Memorial Hospital, Linkou Branch, Taoyuan 333, Taiwan.
Wen-Hung ChungDepartment of Dermatology, Chang Gung Memorial Hospital, Linkou Branch, Taoyuan 333, Taiwan.
Shun-Fa YangInstitute of Medicine, Chung Shan Medical University, Taichung 402, Taiwan.ORCID 0000-0002-0365-7927
Shih-Chi SuWhole-Genome Research Core Laboratory of Human Diseases, Chang Gung Memorial Hospital, Keelung 204, Taiwan.

Funding

Linkou Chang Gung Memorial Hospital BMRPE97Ministry of Science and Technology MOST 109-2314-B-182A-012, MOST 110-2314-B-182A-104, MOST111-2314-B-182A-116
6 · The paper itself

Abstract

Alterations in the gut microbiota composition and their associated metabolic dysfunction exist in psoriasis. However, the impact of biologics on shaping gut microbiota is not well known. This study aimed to determine the association of gut microorganisms and microbiome-encoded metabolic pathways with the treatment in patients with psoriasis. A total of 48 patients with psoriasis, including 30 cases who received an IL-23 inhibitor (guselkumab) and 18 cases who received an IL-17 inhibitor (secukinumab or ixekizumab) were recruited. Longitudinal profiles of the gut microbiome were conducted by using 16S rRNA gene sequencing. The gut microbial compositions dynamically changed in psoriatic patients during a 24-week treatment. The relative abundance of individual taxa altered differently between patients receiving the IL-23 inhibitor and those receiving the IL-17 inhibitor. Functional prediction of the gut microbiome revealed microbial genes related to metabolism involving the biosynthesis of antibiotics and amino acids were differentially enriched between responders and non-responders receiving IL-17 inhibitors, as the abundance of the taurine and hypotaurine pathway was found to be augmented in responders treated with the IL-23 inhibitor. Our analyses showed a longitudinal shift in the gut microbiota in psoriatic patients after treatment. These taxonomic signatures and functional alterations of the gut microbiome could serve as potential biomarkers for the response to biologics treatment in psoriasis.

Indexed as

Biological ProductsGastrointestinal MicrobiomePsoriasisHumansInterleukin-17Interleukin-23RNA, Ribosomal, 16SBiological ProductsInterleukin-17Interleukin-23RNA, Ribosomal, 16Sguselkumabgut microbiotainterleukin-17 inhibitorinterleukin-23 inhibitorixekizumabmetabolic pathwaypsoriasissecukinumab

Identifiers

PMID36902001
PMCPMC10002560

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.