Evidence mapPaperPMID 36926610Full record

ArticleJournal of ginseng research2023

Protective effect and mechanism of ginsenoside Rg2 on atherosclerosis.

Qianqian Xue, Tao Yu, Zhibin Wang, Xiuxiu Fu, Xiaoxin Li, Lu Zou, Min Li, Jae Youl Cho, Yanyan Yang

Open access · goldAbstract read
In one paragraph

Article in Journal of ginseng research, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed
3.3field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

20 citing papers in PubMed, 40 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. Article
  5. Frontiers in cardiovascular medicine · 2026
    Article
  6. Article
  7. Review
  8. Journal of ginseng research · 2025
    Review
  9. Ginsenoside Rg2, a principal effective ingredient ofJournal of ginseng research · 2025
    Article
  10. Article
  11. Review
  12. Article
  13. Article
  14. Article
  15. Article
  16. Review
  17. Review
  18. Therapeutic Applications of Ginseng Natural Compounds for Health Management.International journal of molecular sciences · 2023
    Review
  19. Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 2 institutions in 2 countries.

Qianqian XueDepartment of Pharmacology, School of Pharmacy, Qingdao University, China.
Tao YuInstitute for translational medicine, The Affiliated Hospital of Qingdao University, China.
Zhibin WangDepartment of Cardiac Ultrasound, The Affiliated Hospital of Qingdao University, China.
Xiuxiu FuDepartment of Cardiac Ultrasound, The Affiliated Hospital of Qingdao University, China.
Xiaoxin LiInstitute for translational medicine, The Affiliated Hospital of Qingdao University, China.
Lu ZouInstitute for translational medicine, The Affiliated Hospital of Qingdao University, China.
Min LiInstitute for translational medicine, The Affiliated Hospital of Qingdao University, China.
Jae Youl ChoDepartment of Integrative Biotechnology and Biomedical Institute for Convergence at SKKU (BICS), Sungkyunkwan University, Suwon, Republic of Korea.
Yanyan YangDepartment of Immunology, Basic Medicine School, Qingdao University, China.
Qingdao University · CNSungkyunkwan University · KR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: Ginsenoside Rg2 (Rg2) has a variety of pharmacological activities and provides benefits during inflammation, cancer, and other diseases. However, there are no reports about the relationship between Rg2 and atherosclerosis. Methods: We used 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) to detect the cell viability of Rg2 in vascular smooth muscle cells (VSMCs) and human umbilical vein endothelial cells (HUVECs). The expression of inflammatory factors in HUVECs and the expression of phenotypic transformation-related marker in VSMCs were detected at mRNA levels. Western blot method was used to detect the expression of inflammation pathways and the expression of phenotypic transformation at the protein levels. The rat carotid balloon injury model was performed to explore the effect of Rg2 on inflammation and phenotypic transformation Results: Rg2 decreased the expression of inflammatory factors induced by lipopolysaccharide in HUVECs-without affecting cell viability. These events depend on the blocking regulation of NF-κB and p-ERK signaling pathway. In VSMCs, Rg2 can inhibit the proliferation, migration, and phenotypic transformation of VSMCs induced by platelet derived growth factor-BB (PDGF-BB)-which may contribute to its anti-atherosclerotic role. In rats with carotid balloon injury, Rg2 can reduce intimal proliferation after injury, regulate the inflammatory pathway to reduce inflammatory response, and also suppress the phenotypic transformation of VSMCs. Conclusion: These results suggest that Rg2 can exert its anti-atherosclerotic effect at the cellular level and animal level, which provides a more sufficient basis for ginseng as a functional dietary regulator.

Indexed as

atherosclerosisendothelial cellginsenoside Rg2inflammationsignaling pathway

Identifiers

PMID36926610
PMCPMC10014178
OpenAlexW4291632782

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.