Evidence map›Paper›PMID 36977674›Full record

ArticleCell death & disease2023

Mitochondria-derived H

Hua Bai, Cong-Wen Fang, Ying Shi, Song Zhai, An Jiang, Ying-Na Li, Lin Wang, Qi-Ling Liu, Geng-Yao Zhou, Jia-Hao Cao and 3 more

Open access · goldFull text read
In one paragraph

Article in Cell death & disease, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
4.0field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 27 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors at 7 institutions in 1 country.

Hua Bai *Frontiers Science Center for Flexible Electronics, Xi'an Institute of Flexible Electronics (IFE) and Xi'an Institute of Biomedical Materials & Engineering, Northwestern Polytechnical University, Xi'an, 710072, China.ORCID 0000-0002-7974-4816
Cong-Wen Fang *Department of Nutrition and Food Hygiene, School of Preventive Medicine, The Fourth Military Medical University, Xi'an, 710032, China.ORCID 0000-0003-4944-0763
Ying Shi *School of Public Health, Shaanxi University of Chinese Medicine, Xianyang, 712046, China.ORCID 0000-0002-7815-6449
Song ZhaiDepartment of Infectious Diseases, The Second Affiliated Hospital, Xi'an Jiaotong University, Xi'an, 710004, China.
An JiangDepartment of General Surgery Ward 4, The Second Affiliated Hospital, Xi'an Jiaotong University, Xi'an, 710004, China.
Ying-Na LiPeople's Hospital of Zhuhai High-tech Industrial Development Zone, Zhuhai, 519085, China.ORCID 0000-0002-8374-0201
Lin WangDepartment of Hepatobiliary Surgery, Xijing Hospital, The Fourth Military Medical University, Xi'an, 710032, China.ORCID 0000-0001-5586-3012
Qi-Ling LiuSchool of Public Health, Shaanxi University of Chinese Medicine, Xianyang, 712046, China.ORCID 0000-0002-3479-5601
Geng-Yao ZhouDepartment of Nutrition and Food Hygiene, School of Preventive Medicine, The Fourth Military Medical University, Xi'an, 710032, China.ORCID 0000-0002-5921-018X
Jia-Hao CaoSchool of Public Health, Shaanxi University of Chinese Medicine, Xianyang, 712046, China.
Jia LiKey Laboratory of Aerospace Medicine of Ministry of Education, School of Aerospace Medicine, The Fourth Military Medical University, Xi'an, 710032, China. jiali816@fmmu.edu.cn.ORCID 0000-0001-9492-6346
Xue-Kang YangDepartment of Burns and Cutaneous Surgery, Xijing Hospital, The Fourth Military Medical University, Xi'an, 710032, China. yangxuekangburns@163.com.ORCID 0000-0002-7318-8927
Xu-Jun QinKey Laboratory for Space Biosciences and Biotechnology, School of Life Sciences, Northwestern Polytechnical University, Xi'an, 710072, China. qinxujun@nwpu.edu.cn.ORCID 0000-0002-2529-1946
Air Force Medical University · CNNorthwestern Polytechnical University · CNSecond Affiliated Hospital of Xi'an Jiaotong University · CNShaanxi University of Chinese Medicine · CNXijing Hospital · CNXian Center for Disease Control and Prevention · CNZhuhai People's Hospital · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Reactive oxygen species (ROS) can induce oxidative injury and are generally regarded as toxic byproducts, although they are increasingly recognized for their signaling functions. Increased ROS often accompanies liver regeneration (LR) after liver injuries, however, their role in LR and the underlying mechanism remains unclear. Here, by employing a mouse LR model of partial hepatectomy (PHx), we found that PHx induced rapid increases of mitochondrial hydrogen peroxide (H

Indexed as

HepatectomyLiver RegenerationAnimalsDisease Models, AnimalForkhead Box Protein O3Hydrogen PeroxideMiceMitochondriaReactive Oxygen SpeciesSignal TransductionForkhead Box Protein O3FoxO3 protein, mouseHydrogen PeroxideReactive Oxygen Species

Identifiers

PMID36977674
PMCPMC10050396
OpenAlexW4361204536

What Socratic holds

Textfull text, public
LicenceCC BY
measurements read43
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.