Evidence mapPaperPMID 37154159Full record

ArticleJCI insight2023

Aging alters mechanisms underlying voluntary movements in spinal motor neurons of mice, primates, and humans.

Ryan W Castro, Mikayla C Lopes, Robert E Settlage, Gregorio Valdez

Open access · goldAbstract read
In one paragraph

Article in JCI insight, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
2.5field-weighted citation impact, top 10% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed, 20 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Article
  6. Tongue blade neuromuscular junction defects in old F344 rats.Respiratory physiology & neurobiology · 2026
    Article
  7. Article
  8. Article
  9. Review
  10. Sex and Age-Related Changes in Lean Mass of C57BL/6 Mice: A New Method for Assessing Body Composition.Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE Engineering in Medicine and Biology Society. Annual International Conference · 2025
    Article
  11. Article
  12. Article
  13. Article
  14. Article
  15. Article
  16. Article
  17. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 2 countries.

Ryan W CastroNeuroscience Graduate Program.
Mikayla C LopesDepartment of Molecular Biology, Cellular Biology, and Biochemistry.
Robert E SettlageDepartment of Advanced Research Computing, Virginia Tech, Blacksburg, Virginia, USA.
Gregorio ValdezDepartment of Molecular Biology, Cellular Biology, and Biochemistry.
Allen Institute for Brain Science · USVirginia Tech · US

Funding

NIA NIH HHS F99 AG068442NIA NIH HHS R01 AG055545NIA NIH HHS R56 AG051501NINDS NIH HHS R21 NS106313
6 · The paper itself

Abstract

Spinal motor neurons have been implicated in the loss of motor function that occurs with advancing age. However, the cellular and molecular mechanisms that impair the function of these neurons during aging remain unknown. Here, we show that motor neurons do not die in old female and male mice, rhesus monkeys, and humans. Instead, these neurons selectively and progressively shed excitatory synaptic inputs throughout the soma and dendritic arbor during aging. Thus, aged motor neurons contain a motor circuitry with a reduced ratio of excitatory to inhibitory synapses that may be responsible for the diminished ability to activate motor neurons to commence movements. An examination of the motor neuron translatome (ribosomal transcripts) in male and female mice reveals genes and molecular pathways with roles in glia-mediated synaptic pruning, inflammation, axonal regeneration, and oxidative stress that are upregulated in aged motor neurons. Some of these genes and pathways are also found altered in motor neurons affected with amyotrophic lateral sclerosis (ALS) and responding to axotomy, demonstrating that aged motor neurons are under significant stress. Our findings show mechanisms altered in aged motor neurons that could serve as therapeutic targets to preserve motor function during aging.

Indexed as

Amyotrophic Lateral SclerosisMotor NeuronsAgedAgingAnimalsFemaleHumansMaleMicePrimatesSynapsesAgingMovement disordersNeurodegenerationNeuromuscular diseaseNeuroscience

Identifiers

PMID37154159
PMCPMC10243831
OpenAlexW4375954959

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.