Evidence mapPaperPMID 37192005Full record

ArticleJCI insight2023

The glucagon-like peptide-1 (GLP-1) analogue semaglutide reduces alcohol drinking and modulates central GABA neurotransmission.

Vicky Chuong, Mehdi Farokhnia, Sophia Khom, Claire L Pince, Sophie K Elvig, Roman Vlkolinsky, Renata Cn Marchette, George F Koob, Marisa Roberto, Leandro F Vendruscolo and 1 more

Registry-linked trialOpen access · goldAbstract read
In one paragraph

Article in JCI insight, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT06015893 (Semaglutide Therapy for Alcohol Reduction), which is not on this map. Cited by 103 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
103citing papers in PubMed, 2 pooled it
26.2field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT06015893 phase2active not recruitingnot on this map

Semaglutide Therapy for Alcohol Reduction (STAR): A Proof-of-Concept Phase II Clinical Trial

TypeinterventionalSponsorNational Institute on Drug Abuse (NIDA)Ran2023 to 2027Enrolled63ConditionsAddiction, Alcohol Use DisorderArmsTake Control, Semaglutide
3 · Its place in the literature

Who cites it

103 citing papers in PubMed, 2 syntheses or guidelines pooled it, 176 citations in OpenAlex.

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43 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 4 institutions in 2 countries.

Vicky ChuongClinical Psychoneuroendocrinology and Neuropsychopharmacology Section, Translational Addiction Medicine Branch, National Institute on Drug Abuse Intramural Research Program (NIDA IRP) and National Institute on Alcohol Abuse and Alcoholism Division of Intramural Clinical and Biological Research (NIAAA DICBR), NIH, Baltimore and Bethesda, Maryland, USA.
Mehdi FarokhniaClinical Psychoneuroendocrinology and Neuropsychopharmacology Section, Translational Addiction Medicine Branch, National Institute on Drug Abuse Intramural Research Program (NIDA IRP) and National Institute on Alcohol Abuse and Alcoholism Division of Intramural Clinical and Biological Research (NIAAA DICBR), NIH, Baltimore and Bethesda, Maryland, USA.
Sophia KhomDepartment of Molecular Medicine, The Scripps Research Institute, La Jolla, California, USA.
Claire L PinceClinical Psychoneuroendocrinology and Neuropsychopharmacology Section, Translational Addiction Medicine Branch, National Institute on Drug Abuse Intramural Research Program (NIDA IRP) and National Institute on Alcohol Abuse and Alcoholism Division of Intramural Clinical and Biological Research (NIAAA DICBR), NIH, Baltimore and Bethesda, Maryland, USA.
Sophie K ElvigNeurobiology of Addiction Section, NIDA IRP, NIH, Baltimore, Maryland, USA.
Roman VlkolinskyDepartment of Molecular Medicine, The Scripps Research Institute, La Jolla, California, USA.
Renata Cn MarchetteNeurobiology of Addiction Section, NIDA IRP, NIH, Baltimore, Maryland, USA.
George F KoobNeurobiology of Addiction Section, NIDA IRP, NIH, Baltimore, Maryland, USA.
Marisa RobertoDepartment of Molecular Medicine, The Scripps Research Institute, La Jolla, California, USA.
Leandro F VendruscoloStress and Addiction Neuroscience Unit, NIDA IRP and National Institute on Alcohol Abuse and Alcoholism Division of Intramural Clinical and Biological Research, National Institutes of Health, Baltimore, Maryland, USA.
Lorenzo LeggioClinical Psychoneuroendocrinology and Neuropsychopharmacology Section, Translational Addiction Medicine Branch, National Institute on Drug Abuse Intramural Research Program (NIDA IRP) and National Institute on Alcohol Abuse and Alcoholism Division of Intramural Clinical and Biological Research (NIAAA DICBR), NIH, Baltimore and Bethesda, Maryland, USA.
National Institutes of Health · USScripps Research Institute · USNational Institute on Drug Abuse · USNational Institute on Alcohol Abuse and Alcoholism · US

Funding

Neurobiology of AddictionZIADA000602 · NATIONAL INSTITUTE ON DRUG ABUSE · 2025 to 2025
$2.8M
Clinical Psychoneuroendocrinology and Neuropsychopharmacology (CPN)ZIADA000635 · NATIONAL INSTITUTE ON DRUG ABUSE · 2025 to 2025
$2.7M
Electrophysiology of alcohol in extended amygdalaU01AA013498 · SCRIPPS RESEARCH INSTITUTE, THE · 2001 to 2025
$2.1M
Stress and Addiction Neuroscience UnitZIADA000644 · NATIONAL INSTITUTE ON DRUG ABUSE · 2025 to 2025
$1.7M
Neuroplasticity of the Extended Amygdala CRF circuitry in alcohol dependenceR01AA021491 · NIAAA · SCRIPPS RESEARCH INSTITUTE, THE · PI MARISA ROBERTO · 2022 to 2024
$1.2M
Intramural NIH HHS ZIA DA000602Intramural NIH HHS ZIA DA000635Intramural NIH HHS ZIA DA000644NIAAA NIH HHS R01 AA021491NIAAA NIH HHS U01 AA013498
6 · The paper itself

Abstract

Growing evidence indicates that the glucagon-like peptide-1 (GLP-1) system is involved in the neurobiology of addictive behaviors, and GLP-1 analogues may be used for the treatment of alcohol use disorder (AUD). Here, we examined the effects of semaglutide, a long-acting GLP-1 analogue, on biobehavioral correlates of alcohol use in rodents. A drinking-in-the-dark procedure was used to test the effects of semaglutide on binge-like drinking in male and female mice. We also tested the effects of semaglutide on binge-like and dependence-induced alcohol drinking in male and female rats, as well as acute effects of semaglutide on spontaneous inhibitory postsynaptic currents (sIPSCs) from central amygdala (CeA) and infralimbic cortex (ILC) neurons. Semaglutide dose-dependently reduced binge-like alcohol drinking in mice; a similar effect was observed on the intake of other caloric/noncaloric solutions. Semaglutide also reduced binge-like and dependence-induced alcohol drinking in rats. Semaglutide increased sIPSC frequency in CeA and ILC neurons from alcohol-naive rats, suggesting enhanced GABA release, but had no overall effect on GABA transmission in alcohol-dependent rats. In conclusion, the GLP-1 analogue semaglutide decreased alcohol intake across different drinking models and species and modulated central GABA neurotransmission, providing support for clinical testing of semaglutide as a potentially novel pharmacotherapy for AUD.

Indexed as

AlcoholismGlucagon-Like Peptide 1Alcohol DrinkingAnimalsFemalegamma-Aminobutyric AcidMaleMiceRatsSemaglutideSynaptic Transmissiongamma-Aminobutyric AcidGlucagon-Like Peptide 1SemaglutideAddictionEndocrinologyNeuroscience

Identifiers

PMID37192005
PMCPMC10371247
OpenAlexW4376643286

What Socratic holds

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LicenceCC BY
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.