Evidence mapPaperPMID 37209227Full record

ReviewDiabetologia2023

Gut hormone-based pharmacology: novel formulations and future possibilities for metabolic disease therapy.

Matthias Tschöp, Ruben Nogueiras, Bo Ahrén

Open access · hybridAbstract readReview
In one paragraph

Review in Diabetologia, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 39 papers.

0numbers the graph read from it
0cells of the map it votes in
39citing papers in PubMed
14.5field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

39 citing papers in PubMed, 73 citations in OpenAlex.

  1. Trial
  2. Article
  3. Article
  4. The emerging role of branched-chain amino acid metabolism in obesity.International journal of obesity (2005) · 2026
    Review
  5. Review
  6. Article
  7. Glucagon-Like Peptide-1 Receptor Agonists: Their Potential Role in Prediabetes.Diabetes therapy : research, treatment and education of diabetes and related disorders · 2026
    Review
  8. Review
  9. Review
  10. Review
  11. Insights into the Mechanism of Action of Tirzepatide: A Narrative Review.Diabetes therapy : research, treatment and education of diabetes and related disorders · 2026
    Review
  12. Review
  13. Review
  14. Article
  15. Review
  16. Old and new anti-obesity drugs.Journal of diabetes and metabolic disorders · 2025
    Review
  17. Article
  18. Review
  19. Review
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 3 institutions in 3 countries.

Matthias TschöpInstitute for Diabetes and Obesity, Helmholtz Zentrum, München, Germany.ORCID http://orcid.org/0000-0002-4744-371X
Ruben NogueirasDepartment of Physiology, University of Santiago de Compostela, Santiago de Compostela, Spain.ORCID http://orcid.org/0000-0002-9976-9930
Bo AhrénDepartment of Clinical Sciences Lund, Lund University, Lund, Sweden. Bo.Ahren@med.lu.se.ORCID http://orcid.org/0000-0002-9804-5340
Helmholtz Zentrum München · DELund University · SEUniversidade de Santiago de Compostela · ES

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Glucagon-like peptide-1 (GLP-1) receptor agonists are established pharmaceutical therapies for the treatment of type 2 diabetes and obesity. They mimic the action of GLP-1 to reduce glucose levels through stimulation of insulin secretion and inhibition of glucagon secretion. They also reduce body weight by inducing satiety through central actions. The GLP-1 receptor agonists used clinically are based on exendin-4 and native GLP-1 and are available as formulations for daily or weekly s.c. or oral administration. GLP-1 receptor agonism is also achieved by inhibitors of dipeptidyl peptidase-4 (DPP-4), which prevent the inactivation of GLP-1 and glucose-dependent insulinotropic polypeptide (GIP), thereby prolonging their raised levels after meal ingestion. Other developments in GLP-1 receptor agonism include the formation of small orally available agonists and compounds with the potential to pharmaceutically stimulate GLP-1 secretion from the gut. In addition, GLP-1/glucagon and GLP-1/GIP dual receptor agonists and GLP-1/GIP/glucagon triple receptor agonists have shown the potential to reduce blood glucose levels and body weight through their effects on islets and peripheral tissues, improving beta cell function and stimulating energy expenditure. This review summarises developments in gut hormone-based therapies and presents the future outlook for their use in type 2 diabetes and obesity.

Indexed as

Diabetes Mellitus, Type 2GlucagonBody WeightGlucagon-Like Peptide 1Glucagon-Like Peptide-1 Receptor AgonistsGlucoseHumansObesityGlucagonGlucagon-Like Peptide 1Glucagon-Like Peptide-1 Receptor AgonistsGlucoseDiabetesDouble receptor agonistsGIPGLP-1 receptor agonistsGlucagonObesityReviewTriple receptor agonists

Identifiers

PMID37209227
PMCPMC10474213
OpenAlexW4377138246

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.