Evidence map›Paper›PMID 37247304›Full record

ArticleAsian Pacific journal of cancer prevention : APJCP2023

The Cytotoxic Effect of Thymoquinone Enhance on HepG2 Cell Line due to Induction of Fenton Reaction by Hydrogen Peroxide: An In Vitro and In Silico Study.

Sara Ghelichkhani, Javad Saffari-Chaleshtori, Fatemeh Ghaffari, Amir Nili-Ahmadabadi

Open access · goldAbstract read
In one paragraph

Article in Asian Pacific journal of cancer prevention : APJCP, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
2.0field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 5 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 1 country.

Sara GhelichkhaniMedicinal Plants and Natural Products Research Center, Hamadan University of Medical Sciences, Hamadan, Iran.
Javad Saffari-ChaleshtoriClinical Biochemistry Research Center, Basic Health Sciences Institute, Shahrekord University of Medical Sciences, Shahrekord, Iran.
Fatemeh GhaffariMedicinal Plants and Natural Products Research Center, Hamadan University of Medical Sciences, Hamadan, Iran.
Amir Nili-AhmadabadiMedicinal Plants and Natural Products Research Center, Hamadan University of Medical Sciences, Hamadan, Iran.
Hamedan University of Medical Sciences · IRShahrekord University of Medical Sciences · IR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

objectiveThymoquinone (TQ) is a component derived from the volatile oil of Nigella sativa. Fenton reaction induction is a well-known strategy to prevent the growth of cancer cells which can stimulate by hydrogen peroxide. This study was designed to investigate the TQ effects on hydrogen peroxide-induced cytotoxicity.

methodsIn this study, HepG2 cell survival, reactive oxygen species (ROS) production, cell membrane integrity, and changes of superoxide dismutase (SOD)/ catalase (CAT) activity were evaluated following incubation of HepG2 cells with 31 μM hydrogen peroxide and different concentrations of TQ (18.5, 37 and 75 μM). In addition, molecular docking studies on the interference of TQ with CAT/SOD enzymes were investigated.

resultsOur findings showed that TQ low concentration can increase the survival of HepG2 cells when exposed to hydrogen peroxide, and on the contrary, its high concentration can potentiate cytotoxicity induced by hydrogen peroxide. The TQ alongside hydrogen peroxide increased the production of ROS, which was related to increase CAT and SOD activity in the HepG2 cells. Molecular docking findings showed that TQ effects on the formation of free radicals were not related to its chemical interference with the structure of the SOD/CAT molecules.

conclusionFenton reaction induction may increase the effectiveness of TQ in preventing HepG2 cells proliferation.

Indexed as

Antineoplastic AgentsHydrogen PeroxideAntioxidantsBenzoquinonesHep G2 CellsHumansMolecular Docking SimulationOxidative StressReactive Oxygen SpeciesSuperoxide DismutaseAntineoplastic AgentsAntioxidantsBenzoquinonesHydrogen PeroxideReactive Oxygen SpeciesSuperoxide DismutasethymoquinoneCatalaseHydrogen peroxideoxidative stressSuperoxide dismutasethymoquinone

Identifiers

PMID37247304
PMCPMC10495912
OpenAlexW4378714645

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.